Document Detail


Local cerebral glucose utilization decreases after heatstroke onset in rats.
MedLine Citation:
PMID:  11479024     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The quantitative autoradiographic 2-[(14)C]-deoxy-D-glucose methods were used to assess the local cerebral glucose utilization (LCGU) in 46 brain structures in rats suffering from heatstroke. Heatstroke was induced by exposing the animals to an ambient temperature of 42 degrees C. The time at which the local cerebral blood flow (CBF) and mean arterial pressure (MAP) decreased from their peak levels was taken as the onset of heatstroke. Control rats were exposed to a temperature of 24 degrees C. The values of local CBF, MAP and means of total LCGU after heatstroke onset were all significantly lower than those in control rats. However, the values of colonic temperature were greater. Thus, it appears that the decreased cerebral metabolism and perfusion can be due to secondary effect of hypertension after heatstroke onset.
Authors:
L Chang; Y Huang; M Lin
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Neuroscience letters     Volume:  308     ISSN:  0304-3940     ISO Abbreviation:  Neurosci. Lett.     Publication Date:  2001 Aug 
Date Detail:
Created Date:  2001-07-31     Completed Date:  2001-09-20     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7600130     Medline TA:  Neurosci Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  206-8     Citation Subset:  IM    
Affiliation:
Institute of Physiology, National Yang-Ming University, Taipei, Taiwan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Carbon Radioisotopes / diagnostic use
Cerebral Cortex / blood supply,  metabolism*
Cerebrovascular Circulation
Deoxyglucose / pharmacokinetics
Glucose / metabolism*
Heat Stroke / metabolism*
Hypotension / metabolism
Male
Rats
Rats, Sprague-Dawley
Chemical
Reg. No./Substance:
0/Carbon Radioisotopes; 154-17-6/Deoxyglucose; 50-99-7/Glucose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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