Document Detail


Liquorice-induced sodium retention. Merely an acquired condition of apparent mineralocorticoid excess? A case report.
MedLine Citation:
PMID:  11202632     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Excessive ingestion of liquorice may result in sodium retention, hypertension, hypokalemia, and suppression of renin and aldosterone. Similarities between liquorice-induced effects and congenital apparent mineralocorticoid excess have recently been emphasized, as in both conditions, reduced activity of the enzyme 11 beta-hydroxysteroid dehydrogenase type 2 allows cortisol to act as a potent mineralocorticoid. We report a case of generalized edema without any increase in blood pressure, with biochemical and hormonal features of apparent mineralocorticoid excess, in a young woman who had been ingesting substantial amounts of liquorice for several years. Liquorice-induced wide-spread edema without hypertension in our patient, as well as in a few other cases previously reported, and the more common occurrence of edema associated with hypertension challenge the current explanation of liquorice syndrome as a purely acquired apparent mineralocorticoid excess. Indeed, in both congenital apparent and true mineralocorticoid excess, edema is typically absent, as a result of the sodium escape phenomenon. As pressure-natriuresis may be an essential mechanism accounting for the sodium escape phenomenon, some component of liquorice could partially or completely oppose the circulatory response that converts liquorice-induced sodium retention into blood pressure elevation. In patients with unexplained generalized edema and hypokalemia without hypertension, liquorice ingestion should be carefully investigated and the renin-aldosterone system should be assayed.
Authors:
A Negro; E Rossi; G Regolisti; F Perazzoli
Publication Detail:
Type:  Case Reports; Journal Article    
Journal Detail:
Title:  Annali italiani di medicina interna : organo ufficiale della Società italiana di medicina interna     Volume:  15     ISSN:  0393-9340     ISO Abbreviation:  Ann. Ital. Med. Int.     Publication Date:    2000 Oct-Dec
Date Detail:
Created Date:  2001-02-01     Completed Date:  2001-02-22     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  8806705     Medline TA:  Ann Ital Med Int     Country:  Italy    
Other Details:
Languages:  eng     Pagination:  296-300     Citation Subset:  IM    
Affiliation:
II Divisione di Medicina Interna, Azienda Ospedaliera Santa Maria Nuova di Reggio Emilia.
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MeSH Terms
Descriptor/Qualifier:
Adult
Blood Pressure / physiology
Edema / blood,  etiology
Female
Glycyrrhiza / adverse effects*
Humans
Hypernatremia / blood,  etiology*
Mineralocorticoids / metabolism
Plants, Medicinal*
Sodium / urine
Chemical
Reg. No./Substance:
0/Mineralocorticoids; 7440-23-5/Sodium

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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