Document Detail


Lessons from imprinted multilocus loss of methylation in human syndromes: A step toward understanding the mechanisms underlying these complex diseases.
MedLine Citation:
PMID:  20495355     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Genomic imprinting is one of the most important epigenetic mechanisms of regulation. Faithful establishment and maintenance of imprinting during mammalian fetal development is crucial for correct fetal and postnatal development of the individual. In humans, numerous complex syndromes (including Russell Silver Syndrome and Beckwith Wiedemann syndrome) and cancers are associated with loss of imprinting (LOI) at particular loci. Over recent years, there has been increasing evidence that LOI is not only an isolated event occurring at a given locus involved in a particular syndrome, but that many patients with a given syndrome have multilocus imprinting defects affecting both parental alleles. This new evidence demonstrates that these anomalies occur during the post-fertilization period of fetal development and raises the question of what mechanisms lead to these multilocus imprinting defects. Identification of the factors involved in the maintenance and/or the establishment of imprinting is undoubtedly crucial for understanding both the mechanisms underlying imprinting regulation and which disruptions lead to these complex diseases.
Authors:
Salah Azzi; Sylvie Rossignol; Yves Le Bouc; Irène Netchine
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Publication Detail:
Type:  Journal Article     Date:  2010-07-01
Journal Detail:
Title:  Epigenetics : official journal of the DNA Methylation Society     Volume:  5     ISSN:  1559-2308     ISO Abbreviation:  Epigenetics     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-12-14     Completed Date:  2011-04-06     Revised Date:  2011-08-08    
Medline Journal Info:
Nlm Unique ID:  101265293     Medline TA:  Epigenetics     Country:  United States    
Other Details:
Languages:  eng     Pagination:  373-7     Citation Subset:  IM    
Affiliation:
APHP, Hôpital Armand Trousseau, Laboratoire d’Explorations Fonctionnelles Endocriniennes, INSERM UMR-S938 Team 4, Université Pierre et Marie Curie-Paris 6, Paris, France.
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MeSH Terms
Descriptor/Qualifier:
Beckwith-Wiedemann Syndrome / genetics*
DNA Methylation
Epigenesis, Genetic
Genomic Imprinting / genetics*
Humans
Silver-Russell Syndrome / genetics*
Syndrome

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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