Document Detail


Leishmania exosomes modulate innate and adaptive immune responses through effects on monocytes and dendritic cells.
MedLine Citation:
PMID:  20881185     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We investigated the properties of leishmania exosomes with respect to influencing innate and adaptive immune responses. Exosomes from Leishmania donovani modulated human monocyte cytokine responses to IFN-γ in a bimodal fashion by promoting IL-10 production and inhibiting that of TNF-α. Moreover, these vesicles were inhibitory with respect to cytokine responses (IL-12p70, TNF-α, and IL-10) by human monocyte-derived dendritic cells. Exosomes from wild-type (WT) L. donovani failed to prime monocyte-derived dendritic cells to drive the differentiation of naive CD4 T cells into IFN-γ-producing Th1 cells. In contrast, vesicles from heat shock protein (HSP)100(-/-) L. donovani showed a gain-of-function and proinflammatory phenotype and promoted the differentiation of naive CD4 lymphocytes into Th1 cells. Proteomic analysis showed that exosomes from WT and HSP100(-/-) leishmania had distinct protein cargo, suggesting that packaging of proteins into exosomes is dependent in part on HSP100. Treatment of C57BL/6 mice with WT L. donovani exosomes prior to challenge with WT organisms exacerbated infection and promoted IL-10 production in the spleen. In contrast, HSP100(-/-) exosomes promoted spleen cell production of IFN-γ and did not adversely affect hepatic parasite burdens. Furthermore, the proparasitic properties of WT exosomes were not species specific because BALB/c mice exposed to Leishmania major exosomes showed increased Th2 polarization and exacerbation of disease in response to infection with L. major. These findings demonstrate that leishmania exosomes are predominantly immunosuppressive. Moreover, to our knowledge, this is the first evidence to suggest that changes in the protein cargo of exosomes may influence the impact of these vesicles on myeloid cell function.
Authors:
Judith Maxwell Silverman; Joachim Clos; Eva Horakova; Adele Y Wang; Martina Wiesgigl; Isabelle Kelly; Miriam A Lynn; W Robert McMaster; Leonard J Foster; Megan K Levings; Neil E Reiner
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-09-29
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  185     ISSN:  1550-6606     ISO Abbreviation:  J. Immunol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-10-21     Completed Date:  2010-11-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  5011-22     Citation Subset:  AIM; IM    
Affiliation:
Division of Infectious Diseases, Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.
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MeSH Terms
Descriptor/Qualifier:
Adaptive Immunity / immunology*
Animals
Antigens, Protozoan / immunology
Cell Differentiation / immunology
Cell Separation
Cytokines / biosynthesis,  immunology
Dendritic Cells / immunology,  microbiology*
Electrophoresis, Gel, Two-Dimensional
Exosomes / immunology*
Flow Cytometry
Heat-Shock Proteins / immunology,  metabolism
Humans
Immunity, Innate / immunology*
Leishmania donovani / immunology*,  metabolism
Leishmaniasis / immunology
Lymphocyte Activation / immunology
Mice
Mice, Inbred BALB C
Mice, Inbred C57BL
Monocytes / immunology,  microbiology*
Protozoan Proteins / immunology,  metabolism
Reverse Transcriptase Polymerase Chain Reaction
Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization
T-Lymphocytes / cytology,  immunology
Grant Support
ID/Acronym/Agency:
MOP-57834//Canadian Institutes of Health Research; MOP-7399//Canadian Institutes of Health Research; MOP-77688//Canadian Institutes of Health Research; MOP-84582//Canadian Institutes of Health Research
Chemical
Reg. No./Substance:
0/Antigens, Protozoan; 0/ClpB protein, Leishmania; 0/Cytokines; 0/Heat-Shock Proteins; 0/Protozoan Proteins

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