Document Detail

Left ventricular performance after acute myocardial infarction.
MedLine Citation:
PMID:  5212392     Owner:  NLM     Status:  MEDLINE    
Current knowledge concerning the major hemodynamic features of acute myocardial infarction has been reviewed and discussed in relation to present concepts of cardiac pathophysiology. The physical examination provides a great deal of information and new, noninvasive methods promise to supplement the bedside appraisal of left ventricular function. Direct hemodynamic methods of serially monitoring patients with acute myocardial infarction are finding increasing application and recently have added considerably to our understanding of this condition. Certain limitations in the use of the central venous pressure, pulmonary arterial pressure, and cardiac output in appraising left ventricular function have become apparent, but together with direct catheterization of the left ventricle such hemodynamic studies have now provided limited correlations between the clinical picture and various hemodynamic patterns. It is becoming increasingly clear that most of these features, including cardiogenic shock, probably reflect varying degrees of left ventricular failure. These initial findings and interpretations will require confirmation, however, and so far insufficient objective data are available concerning the natural history of acute myocardial infarction and its responses to various forms of therapy. The aims of investigations now being carried in specialized Myocardial Infarction Research Units and other cardiovascular research centers, are to gain such further understanding of the pathophysiology of this disease and to aid in its clinical management by developing accurate indirect monitoring techniques as well as new forms of therapy.
J S Karliner; J Ross
Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Progress in cardiovascular diseases     Volume:  13     ISSN:  0033-0620     ISO Abbreviation:  Prog Cardiovasc Dis     Publication Date:  1971 Jan 
Date Detail:
Created Date:  1986-11-10     Completed Date:  1986-11-10     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0376442     Medline TA:  Prog Cardiovasc Dis     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  374-91     Citation Subset:  AIM; IM; S    
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MeSH Terms
Acidosis / complications
Anoxia / complications
Cardiac Output
Catecholamines / pharmacology
Central Venous Pressure
Coronary Circulation
Heart / drug effects
Heart Function Tests
Heart Rate
Insulin / pharmacology
Mitral Valve Insufficiency / etiology
Myocardial Contraction
Myocardial Infarction / physiopathology*
Oxygen Consumption
Pulmonary Wedge Pressure
Stroke Volume
Vascular Resistance
Grant Support
HE-12373//PHS HHS; PH-43-68-1332/PH/PHPPO CDC HHS
Reg. No./Substance:
0/Catecholamines; 11061-68-0/Insulin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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