| Left ventricular overload caused by collateral pulmonary circulation in chronic pneumopathies-myth or reality? | |
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MedLine Citation:
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PMID: 7249659 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The collateral pulmonary blood flow was measured by the dye dilution method in four patients with chronic obstructive pulmonary disease, in 16 patients with diffuse pulmonary fibrosis, in 15 patients with pulmonary embolism, and in three patients with primary pulmonary hypertension. The authors found that collateral pulmonary circulation was small in the first two groups (0.3 % and 1.7% of the pulmonary blood flow, respectively), absent in primary pulmonary hypertension, and high (14.0% of pulmonary flow) in pulmonary embolism. The magnitude of the bronchopulmonary collateral circulation did not correlate with degree of precapillary pulmonary hypertension, left atrial pressure and left ventricular enddiastolic pressure. It seems that the bronchopulmonary collateral flow does not produce diastolic left ventricular overload in chronic pneumopathies. |
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Authors:
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J Endrys; B Král; J Eliás; A Hamet |
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Publication Detail:
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Type: Journal Article |
Journal Detail:
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Title: Cor et vasa Volume: 23 ISSN: 0010-8650 ISO Abbreviation: Cor Vasa Publication Date: 1981 |
Date Detail:
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Created Date: 1981-09-25 Completed Date: 1981-09-25 Revised Date: 2007-11-15 |
Medline Journal Info:
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Nlm Unique ID: 0372614 Medline TA: Cor Vasa Country: CZECHOSLOVAKIA |
Other Details:
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Languages: eng Pagination: 121-5 Citation Subset: IM |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Anoxia
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complications Chronic Disease Forced Expiratory Volume Heart Ventricles / physiopathology* Humans Hypertension, Pulmonary / complications Lung Diseases / complications*, pathology Lung Diseases, Obstructive / complications Pulmonary Circulation* Pulmonary Embolism / complications Pulmonary Fibrosis / complications Vital Capacity |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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