| Late ventricular potentials and heavy drinking. | |
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MedLine Citation:
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PMID: 9326991 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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OBJECTIVES: To assess the effects of chronic drinking on detection of low amplitude signals, and to determine the relation between late ventricular potentials (LVP) and liver biopsy findings. DESIGN: Prospective study. SETTING: General hospital. PATIENTS: 41 consecutive chronic alcoholics without known pre-existing heart disease. METHODS: About four days after each patient's last alcoholic drink, ECG, echocardiography, signal averaged electrocardiogram, liver biopsy, and blood tests were performed. RESULTS: Twenty eight per cent of patients had evidence of LVP. There was a correlation between the percentage of steatosis of the hepatic biopsy and the amplitude of the last 40 ms of average QRS (P = 0.04), the duration of the terminal low amplitude QRS signal (P = 0.05), and the number of positive criteria of late potentials (P = 0.02). CONCLUSIONS: Chronic drinking sufficient to cause steatosis is associated with positive findings on the signal averaged ECG. |
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Authors:
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G Pochmalicki; M Genest; H Jibril |
Publication Detail:
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Type: Journal Article |
Journal Detail:
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Title: Heart (British Cardiac Society) Volume: 78 ISSN: 1355-6037 ISO Abbreviation: Heart Publication Date: 1997 Aug |
Date Detail:
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Created Date: 1997-10-22 Completed Date: 1997-10-22 Revised Date: 2009-11-18 |
Medline Journal Info:
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Nlm Unique ID: 9602087 Medline TA: Heart Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 163-5 Citation Subset: AIM; IM |
Affiliation:
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Service de Cardiologie, C.H.G. Léon Binet, Provins, France. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Alcoholism
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pathology,
physiopathology* Echocardiography Electrocardiography* Fatty Liver / pathology, physiopathology Female Humans Liver / pathology Male Middle Aged Prospective Studies Signal Processing, Computer-Assisted* Ventricular Dysfunction / pathology, physiopathology* |
| Comments/Corrections | |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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