Document Detail


L-arginine is essential for pancreatic beta-cell functional integrity, metabolism and defence from inflammatory challenge.
MedLine Citation:
PMID:  21784771     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
In this work our aim was to determine whether L arginine (a known insulinotropic amino acid) can promote a shift of β-cell intermediary metabolism favoring glutathione (GSH+GSSG) anti-oxidant responses, stimulus-secretion coupling, and functional integrity. Clonal BRIN-BD11 β cells and mouse islets were cultured for 24h at various L-arginine concentrations (0 to 1.15mmol L-1) in the absence or presence of a pro-inflammatory cytokine cocktail (IL-1β, TNFα, IFNγ). Cells were assessed for viability, insulin secretion, GSH, GSSG, glutamate, NO, superoxide, urea, lactate, and for the consumption of glucose and glutamine. Protein levels of NO synthase (NOS-2), AMPK and the HSP72 were also evaluated. We found that L arginine at 1.15 mmol L-1 attenuated the loss of β-cell viability observed in the presence of pro-inflammatory cytokines. L arginine increased total cellular glutathione and glutamate levels but reduced the GSSG/GSH ratio and glutamate release. The amino acid stimulated glucose consumption in the presence of cytokines while also stimulating AMPK phosphorylation and HSP72 expression. Pro-inflammatory cytokines reduced, by at least 50%, chronic (24h) insulin secretion, an effect partially attenuated by L-arginine. Acute insulin secretion was robustly stimulated by L-arginine but this effect was abolished in the presence of cytokines. We conclude that L-arginine can stimulate β cell insulin secretion, anti-oxidant and protective responses, enabling increased functional integrity of β cells and islets in the presence of pro-inflammatory cytokines. Glucose consumption and intermediary metabolism were increased by L-arginine. These results highlight the importance of L-arginine availability for beta-cells during inflammatory challenge.
Authors:
Mauricio Krause; Nh McClenaghan; Peter R Flatt; Paulo Ivo Homem de Bittencourt; Colin Murphy; Philip Newsholme
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-7-22
Journal Detail:
Title:  The Journal of endocrinology     Volume:  -     ISSN:  1479-6805     ISO Abbreviation:  -     Publication Date:  2011 Jul 
Date Detail:
Created Date:  2011-7-25     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0375363     Medline TA:  J Endocrinol     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Affiliation:
M Krause, Science, Institute of Technology Tallaght, Dublin, Dublin 24, Ireland.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Longitudinal associations of socioeconomic position in childhood and adulthood with decline in lung ...
Next Document:  Drug-induced changes in cortical inhibition in medication overuse headache.