Document Detail


Knockdown of MBP-1 in human prostate cancer cells delays cell cycle progression.
MedLine Citation:
PMID:  16762917     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We have previously shown that MBP-1 acts as a general transcriptional repressor, and forced expression of MBP-1 exerts an anti-proliferative effect on a number of human cancer cells. In this report, we have investigated the role of endogenous MBP-1 in cell growth regulation. For this, we generated human prostate cancer cells (PC3) stably transfected with short hairpin RNA targeting MBP-1. We have observed retarded growth and longer doubling time of MBP-1 knockdown PC3 cells as compared with control mock-transfected PC3 cells. Fluorescence-activated cell sorter analysis suggested that PC3 cells expressing MBP-1-specific small interfering RNA accumulated during G2/M phase of the cell cycle. Further analysis suggested that depletion of MBP-1 was associated with reduction of cyclin A and cyclin B1 expression when compared with that of the control cells. A delayed induction of cyclin A and B1 expression was observed in MBP-1-depleted PC3 cells (PC3-4.2) upon serum stimulation, although the level of expression was much lower than that of control PC3 cells. Supplementation of MBP-1 in PC3-4.2 cells restored cyclin A and cyclin B1 expression. Together, these results suggest that knockdown of MBP-1 in prostate cancer cells perturbs cell proliferation by inhibiting cyclin A and cyclin B1 expression.
Authors:
Asish K Ghosh; Robert Steele; Ratna B Ray
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2006-06-08
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  281     ISSN:  0021-9258     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2006 Aug 
Date Detail:
Created Date:  2006-08-14     Completed Date:  2006-09-25     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  23652-7     Citation Subset:  IM    
Affiliation:
Department of Pathology, Saint Louis University, St. Louis, Missouri 63104, USA.
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MeSH Terms
Descriptor/Qualifier:
Cell Cycle / physiology*
Cell Line
Cell Line, Tumor
Cell Proliferation
Cell Size
Cyclin A / antagonists & inhibitors,  biosynthesis
Cyclin B / antagonists & inhibitors,  biosynthesis
Cyclin B1
Growth Inhibitors / antagonists & inhibitors*,  biosynthesis,  genetics,  physiology
Humans
Male
Nerve Tissue Proteins / antagonists & inhibitors*,  genetics*,  physiology
Prostatic Neoplasms / metabolism*,  pathology*,  prevention & control
RNA Interference / physiology*
Repressor Proteins / antagonists & inhibitors*,  biosynthesis,  genetics,  physiology
Time Factors
Transcription Factors / antagonists & inhibitors*,  genetics*,  physiology
Transfection
Grant Support
ID/Acronym/Agency:
CA52799/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/CCNB1 protein, human; 0/Cyclin A; 0/Cyclin B; 0/Cyclin B1; 0/Growth Inhibitors; 0/MBP protein, human; 0/Nerve Tissue Proteins; 0/Repressor Proteins; 0/Transcription Factors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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