Document Detail


Kir6.2 knockout alters neurotransmitter release in mouse striatum: an in vivo microdialysis study.
MedLine Citation:
PMID:  18524485     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
ATP-sensitive potassium (K-ATP) channels have been demonstrated to play important roles in the brain. In the present study, Kir6.2 knockout (Kir6.2-/-) mice were used to examine the contribution of Kir6.2-containing K-ATP channels to the regulation of neurotransmitter release via in vivo microdialysis studies. The results showed that the extracellular levels of monoamine and amino acid neurotransmitters in Kir6.2-/- mouse striatum were similar to those in Kir6.2+/+ mice under basal conditions. After high K+ (100mM) perfusion, the extracellular levels of DA and amino acids were increased in both genotypes. These increases, however, were significantly lower in Kir6.2-/- mice than those in Kir6.2+/+ mice. Extracellular levels of 3,4-dihydroxyphenylacetic acid (DOPAC), a major metabolite of DA, were increased in Kir6.2-/- mice but decreased in Kir6.2+/+ mice in response to high K+ stimulus. The releases of 4-hydroxy-3-methoxy-phenylacetic acid (HVA) and 5-hydroxyindoleacetic acid (5-HIAA) were attenuated to a similar extent in both mouse genotypes. Taken together, this study provides direct in vivo evidence that Kir6.2-containing K-ATP channels play regulatory roles in neurotransmitter release in the striatum.
Authors:
Xue-Ru Shi; Jing Chang; Jian-Hua Ding; Yi Fan; Xiu-Lan Sun; Gang Hu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-05-14
Journal Detail:
Title:  Neuroscience letters     Volume:  439     ISSN:  0304-3940     ISO Abbreviation:  Neurosci. Lett.     Publication Date:  2008 Jul 
Date Detail:
Created Date:  2008-06-16     Completed Date:  2008-09-26     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7600130     Medline TA:  Neurosci Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  230-4     Citation Subset:  IM    
Affiliation:
Jiangsu Key Laboratory of Neurodegeneration, Department of Pharmacology, Nanjing Medical University, 140 Hanzhong Road, Nanjing, Jiangsu 210029, PR China.
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MeSH Terms
Descriptor/Qualifier:
3,4-Dihydroxyphenylacetic Acid / metabolism
Animals
Corpus Striatum / drug effects,  metabolism*
Dopamine / metabolism
Extracellular Fluid / drug effects,  metabolism
Homovanillic Acid / metabolism
Mice
Mice, Inbred C57BL
Mice, Knockout
Microdialysis / methods
Neurotransmitter Agents / metabolism*
Potassium Channels, Inwardly Rectifying / deficiency*
Potassium Chloride / pharmacology
Chemical
Reg. No./Substance:
0/Kir6.2 channel; 0/Neurotransmitter Agents; 0/Potassium Channels, Inwardly Rectifying; 102-32-9/3,4-Dihydroxyphenylacetic Acid; 306-08-1/Homovanillic Acid; 7447-40-7/Potassium Chloride

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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