Document Detail


It's about time: how input timing is used and not used to create emergent properties in the auditory system.
MedLine Citation:
PMID:  21325525     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The hypothesis for directional selectivity of frequency modulations (FMs) invokes a mechanism with an honored tradition in sensory neurobiology, the relative timing of excitation and inhibition. The proposal is that the timing disparity is created by asymmetrical locations of excitatory tuning and inhibitory sidebands. Thus, cells in which the inhibitory sidebands are tuned to frequencies lower than the excitatory tuning are selective for downward sweeping FMs, because frequencies first generate excitation followed by inhibition. Upward sweeping FMs, in contrast, first evoke inhibition that either leads or is coincident with the excitation and prevents discharges. Here we evaluated FM directional selectivity with in vivo whole-cell recordings from the inferior colliculus of awake bats. From the whole-cell recordings, we derived synaptic conductance waveforms evoked by downward and upward FMs. We then tested the effects of shifting inhibition relative to excitation in a model and found that latency shifts had only minor effects on EPSP amplitudes that were often <1.0 mV/ms shift. However, when the PSPs peaked close to spike threshold, even small changes in latency could cause some cells to fire more strongly to a particular FM direction and thus change its directional selectivity. Furthermore, the effect of shifting inhibition depended strongly on initial latency differences and the shapes of the conductance waveforms. We conclude that "timing" is more than latency differences between excitation and inhibition, and response selectivity depends on a complex interaction between the timing, the shapes, and magnitudes of the excitatory and inhibitory conductances and spike threshold.
Authors:
Joshua X Gittelman; George D Pollak
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  The Journal of neuroscience : the official journal of the Society for Neuroscience     Volume:  31     ISSN:  1529-2401     ISO Abbreviation:  J. Neurosci.     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2011-02-17     Completed Date:  2011-04-01     Revised Date:  2011-09-26    
Medline Journal Info:
Nlm Unique ID:  8102140     Medline TA:  J Neurosci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2576-83     Citation Subset:  IM    
Affiliation:
Section of Neurobiology, Institute for Neuroscience, and Center for Perceptual Systems, Patterson Laboratories, The University of Texas at Austin, Austin, Texas 78712, USA. jxg@mail.utexas.edu
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MeSH Terms
Descriptor/Qualifier:
Acoustic Stimulation / methods
Action Potentials / physiology
Animals
Auditory Pathways / cytology,  physiology*
Biophysics / methods
Chiroptera
Electric Stimulation / methods
Excitatory Postsynaptic Potentials / physiology
Models, Neurological
Neural Inhibition / physiology*
Patch-Clamp Techniques
Reaction Time / physiology
Sensory Receptor Cells / physiology
Statistics as Topic
Time Perception / physiology*
Grant Support
ID/Acronym/Agency:
1F32DC009741/DC/NIDCD NIH HHS; DC007856/DC/NIDCD NIH HHS; F32 DC009741-01/DC/NIDCD NIH HHS; R01 DC007856-05/DC/NIDCD NIH HHS
Comments/Corrections

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