Document Detail


Isolation and characterization of portal branch ligation-stimulated Hmga2-positive bipotent hepatic progenitor cells.
MedLine Citation:
PMID:  21075076     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hepatic stem/progenitor cells are one of several cell sources that show promise for restoration of liver mass and function. Although hepatic progenitor cells (HPCs), including oval cells, are induced by administration of certain hepatotoxins in experimental animals, such a strategy would be inappropriate in a clinical setting. Here, we investigated the possibility of isolating HPCs in a portal branch-ligated liver model without administration of any chemical agents. A non-parenchymal cell fraction was prepared from the portal branch-ligated or non-ligated lobe, and seeded onto plates coated with laminin. Most of the cells died, but a small number were able to proliferate. These proliferating cells were cloned as portal branch ligation-stimulated hepatic cells (PBLHCs) by the limiting dilution method. The PBLHCs expressed cytokeratin19, albumin, and Hmga2. The PBLHCs exhibited metabolic functions such as detoxification of ammonium ions and synthesis of urea on Matrigel-coated plates in the presence of oncostatin M. In Matrigel mixed with type I collagen, the PBLHCs became rearranged into cystic and tubular structures. Immunohistochemical staining demonstrated the presence of Hmga2-positive cells around the interlobular bile ducts in the portal branch-ligated liver lobes. In conclusion, successful isolation of bipotent hepatic progenitor cell clones, PBLHCs, from the portal branch-ligated liver lobes of mice provides the possibility of future clinical application of portal vein ligation to induce hepatic progenitor cells.
Authors:
Hiroshi Sakai; Yoh-ichi Tagawa; Miho Tamai; Hiroaki Motoyama; Shinichiro Ogawa; Junpei Soeda; Takenari Nakata; Shinichi Miyagawa
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-11-12
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  403     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-12-20     Completed Date:  2011-01-20     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  298-304     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Surgery, Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto, Nagano 390-8621, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bile Ducts / growth & development
Cell Proliferation
Cell Separation / methods*
Cells, Cultured
HMGA2 Protein / analysis,  biosynthesis
Hepatocytes / cytology*
Liver Regeneration*
Mice
Mice, Inbred C57BL
Morphogenesis
Stem Cells / chemistry,  cytology*
Chemical
Reg. No./Substance:
0/HMGA2 Protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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