Document Detail

Iron and copper-associated cirrhosis in infants. Acquired metal toxicity or genetic disorder?
MedLine Citation:
PMID:  9891605     Owner:  NLM     Status:  MEDLINE    
Iron and Copper are essential trace elements for growth and development of the human infant but are toxic in excess. Lethal cirrhotic syndromes in the neonate and infant have been associated with both copper and iron overload. The relative importance of underlying genetic susceptibility and acquired excess of iron or copper in the pathogenesis of these chronic metal toxicity states is controversial and both syndromes probably encompass a spectrum of conditions.
G Lockitch
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Clinics in laboratory medicine     Volume:  18     ISSN:  0272-2712     ISO Abbreviation:  Clin. Lab. Med.     Publication Date:  1998 Dec 
Date Detail:
Created Date:  1999-01-28     Completed Date:  1999-01-28     Revised Date:  2006-03-28    
Medline Journal Info:
Nlm Unique ID:  8100174     Medline TA:  Clin Lab Med     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  665-71, viii     Citation Subset:  IM    
Department of Pathology, University of British Columbia, Vancouver, Canada.
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MeSH Terms
Copper* / administration & dosage,  metabolism,  poisoning
Hemochromatosis / complications
Infant, Newborn
Iron* / administration & dosage,  metabolism,  poisoning
Iron Overload / complications
Liver Cirrhosis / etiology*,  genetics*
Reg. No./Substance:
7439-89-6/Iron; 7440-50-8/Copper

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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