Document Detail

Iodine-induced thyroid dysfunction.
MedLine Citation:
PMID:  22820214     Owner:  NLM     Status:  Publisher    
PURPOSE OF REVIEW: To summarize the mechanisms of iodine-induced hypothyroidism and hyperthyroidism, identify the risk factors for thyroid dysfunction following an iodine load, and summarize the major sources of excess iodine exposure. RECENT FINDINGS: Excess iodine is generally well tolerated, but individuals with underlying thyroid disease or other risk factors may be susceptible to iodine-induced thyroid dysfunction following acute or chronic exposure. Sources of increased iodine exposure include the global public health efforts of iodine supplementation, the escalating use of iodinated contrast radiologic studies, amiodarone administration in vulnerable patients, excess seaweed consumption, and various miscellaneous sources. SUMMARY: Iodine-induced thyroid dysfunction may be subclinical or overt. Recognition of the association between iodine excess and iodine-induced hypothyroidism or hyperthyroidism is important in the differential diagnosis of patients who present without a known cause of thyroid dysfunction.
Angela M Leung; Lewis E Braverman
Related Documents :
20664274 - Rantes in exhaled breath condensate of patients with severe persistent allergic asthma ...
8491304 - Effects of acetazolamide on overnight oxygenation and acute mountain sickness in patien...
22259654 - Idiopathic chronic urticaria and thyroid autoimmunity: experience of a single center.
3622024 - Total respiratory resistance and reactance in patients with upper airway obstruction.
16498034 - Visceral and testicular calcifications as part of the phenotype in pseudoxanthoma elast...
22412904 - Non-coding keratin variants associate with liver fibrosis progression in patients with ...
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2012-7-19
Journal Detail:
Title:  Current opinion in endocrinology, diabetes, and obesity     Volume:  -     ISSN:  1752-2978     ISO Abbreviation:  -     Publication Date:  2012 Jul 
Date Detail:
Created Date:  2012-7-23     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101308636     Medline TA:  Curr Opin Endocrinol Diabetes Obes     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Section of Endocrinology, Diabetes, and Nutrition, Boston University School of Medicine, Boston, Massachusetts, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

Previous Document:  The propylthiouracil dilemma.
Next Document:  Are Older Adults Who Volunteer to Participate in an Exercise Study Fitter and Healthier than Non-Vol...