Document Detail


Involvement of extracellular superoxide dismutase in regulating brain blood flow.
MedLine Citation:
PMID:  20033309     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The physiological role of extracellular superoxide dismutase (SOD3) has received insufficient study. We investigated the hypothesis that SOD3, which neutralizes superoxide anions (O2(-)) in the intercellular space of the brain, prevents the inactivation of nitric oxide (NO) and is thus involved in regulating cerebral vascular tone. Local brain blood flow was measured in the striatum of anesthetized rats during administration of various combinations of a SOD mimetic, a SOD inhibitor, an NO donor, and an NOS inhibitor into the striatum using a Hamilton syringe. In normal conditions, SOD3 was found to minimize O2(-) levels, protecting endogenously produced NO at a sufficient level to maintain cerebral vascular tone and reactivity. SOD3 was found to increase the vasodilatory effect of endogenously produced NO in the brain. SOD3 was found to neutralize superoxide anions produced in the brain during respiration of 100% O2 and to maintain basal NO levels and its vasodilatory potential in normobaric hyperoxia.
Authors:
I T Demchenko; D R Gutsaeva; A N Moskvin; S Yu Zhilyaev
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-12-22
Journal Detail:
Title:  Neuroscience and behavioral physiology     Volume:  40     ISSN:  1573-899X     ISO Abbreviation:  Neurosci. Behav. Physiol.     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-01-21     Completed Date:  2010-03-22     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0330471     Medline TA:  Neurosci Behav Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  173-8     Citation Subset:  IM    
Affiliation:
I. M. Sechenov Institute of Evolutionary Physiology and Biochemistry, 44 M. Torez Prospekt, 194223 St. Petersburg, Russia. jil@iephb.ru
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Vessels / drug effects,  enzymology,  physiology
Brain / blood supply*,  drug effects,  physiology*
Cerebrovascular Circulation / drug effects,  physiology*
Corpus Striatum / blood supply,  drug effects,  physiology
Enzyme Inhibitors / pharmacology
Extracellular Space / drug effects,  physiology
Hyperoxia / metabolism
NG-Nitroarginine Methyl Ester / pharmacology
Nitric Oxide / metabolism
Nitric Oxide Donors / pharmacology
Nitric Oxide Synthase / antagonists & inhibitors,  metabolism
Rats
S-Nitroso-N-Acetylpenicillamine / pharmacology
Superoxide Dismutase / antagonists & inhibitors,  metabolism*
Superoxides / metabolism
Vasodilation / drug effects,  physiology
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Nitric Oxide Donors; 10102-43-9/Nitric Oxide; 11062-77-4/Superoxides; 50903-99-6/NG-Nitroarginine Methyl Ester; 79032-48-7/S-Nitroso-N-Acetylpenicillamine; EC 1.14.13.39/Nitric Oxide Synthase; EC 1.15.1.1/Sod3 protein, rat; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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