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Investigations on Alternate Approach to Target Mannose Receptors on Macrophages using 4-Sulfated N-Acetyl Galactosamine more Efficiently as Compared to Mannose Decorated Liposomes : An Application in Drug Delivery.
MedLine Citation:
PMID:  21782778     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
In the present study the targeting potential of two different ligands i.e palmitoyl mannose (Man-Lip) and 4-SO(4)GalNAc (Sulf-Lip) to resident macrophages have been investigated after having decorated on the surface of Amphotericin B (AmB) loaded liposomes. In case of Sulf-Lip, the 4-SO(4)GalNAc was adsorbed through electrostatic interaction on cationic liposomes, which was confirmed by change in zeta potential from +48.2 ± 3.7 mV for Lip to +12.2 ± 1.3 mV for Sulf-Lip. The mean particle size of Sulf-Lip and Man-Lip was found to be 139.4 ± 7.4 nm and 147.4 ± 8.6 nm respectively. Flow cytometric data reveals enhanced uptake of Sulf-Lip in both J774 and RAW cell lines as compared to Man-Lip. Intracellular localization studies indicate that the fluorescence intensity of Sulf-Lip was much higher as compared to Man-Lip and Lip formulations. Sulf-Lip and Man-Lip showed significantly higher localization of AmB at all time points compared to Lip (P < .05) after i.v. administration. The studies provides evidences that 4-SO(4)GalNAc possess promising feature for targeting resident macrophages and its application in the conditions of leishmaniasis is in offing.
Authors:
Deepak Singodia; Ashwni Verma; Rahul K Verma; Prabhat Ranjan Mishra
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Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-7-20
Journal Detail:
Title:  Nanomedicine : nanotechnology, biology, and medicine     Volume:  -     ISSN:  1549-9642     ISO Abbreviation:  -     Publication Date:  2011 Jul 
Date Detail:
Created Date:  2011-7-25     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101233142     Medline TA:  Nanomedicine     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Copyright Information:
Copyright © 2011. Published by Elsevier Inc.
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