Document Detail


Intrinsic control of precise dendritic targeting by an ensemble of transcription factors.
MedLine Citation:
PMID:  17276922     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Proper information processing in neural circuits requires establishment of specific connections between pre- and postsynaptic neurons. Targeting specificity of neurons is instructed by cell-surface receptors on the growth cones of axons and dendrites, which confer responses to external guidance cues. Expression of cell-surface receptors is in turn regulated by neuron-intrinsic transcriptional programs. In the Drosophila olfactory system, each projection neuron (PN) achieves precise dendritic targeting to one of 50 glomeruli in the antennal lobe. PN dendritic targeting is specified by lineage and birth order , and their initial targeting occurs prior to contact with axons of their presynaptic partners, olfactory receptor neurons. We search for transcription factors (TFs) that control PN-intrinsic mechanisms of dendritic targeting. We previously identified two POU-domain TFs, acj6 and drifter, as essential players. After testing 13 additional candidates, we identified four TFs (LIM-homeodomain TFs islet and lim1, the homeodomain TF cut, and the zinc-finger TF squeeze) and the LIM cofactor Chip that are required for PN dendritic targeting. These results begin to provide insights into the global strategy of how an ensemble of TFs regulates wiring specificity of a large number of neurons constituting a neural circuit.
Authors:
Takaki Komiyama; Liqun Luo
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Current biology : CB     Volume:  17     ISSN:  0960-9822     ISO Abbreviation:  Curr. Biol.     Publication Date:  2007 Feb 
Date Detail:
Created Date:  2007-02-05     Completed Date:  2007-06-04     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  9107782     Medline TA:  Curr Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  278-85     Citation Subset:  IM    
Affiliation:
Howard Hughes Medical Institute, Department of Biological Sciences, Neurosciences Program, Stanford University, Stanford, California 94305, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Dendrites / genetics,  metabolism*
Drosophila / genetics*,  metabolism
Drosophila Proteins / metabolism*
Homeodomain Proteins / metabolism
Nerve Net / metabolism*
Neurons / metabolism
Olfactory Receptor Neurons / metabolism
Transcription Factors / metabolism*
Zinc Fingers / genetics
Grant Support
ID/Acronym/Agency:
R01-DC005982/DC/NIDCD NIH HHS
Chemical
Reg. No./Substance:
0/Drosophila Proteins; 0/Homeodomain Proteins; 0/Transcription Factors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Male-killing bacteria trigger a cycle of increasing male fatigue and female promiscuity.
Next Document:  AKT-1 regulates DNA-damage-induced germline apoptosis in C. elegans.