Document Detail


Intravenous neridronate for skeletal damage treatment in patients with multiple myeloma.
MedLine Citation:
PMID:  17172186     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Almost 70-80% of the patients with Multiple Myeloma (MM) in advancer phase, of the disease show osteolytic lesions and/or pathologic fractures, with or without secondary osteoporosis. An accelerated osteoclast-mediated bone absorption is believed to be the main cause of bone damage in MM. Osteoclast can be activated by a variety of microenvironmental factors. Bisphosphonates (BF) induce the apoptosis of osteoclasts and inhibit osteoclastogenesis, thus preventing bone absorption. As well as BFs, the so-called second-generation BF (N-BF) may impair the activity of osteoclast. Neridronic acid (NER) is a N-BF molecule officially registered for the treatment of osteogenesis imperfecta. Nevertheless, NER has shown a remarkable efficacy in Paget's disease, postmenopausal osteoporosis and, most recently, in androgen deprivation-treated prostatic carcinoma. The primary endpoint of this study was to evaluate hip and spine Bone Mineral Density (BMD) modifications over the 12-month treatment with NER in a group of patients affected by MM with evidence of initial skeletal damage. Secondary endpoints were (1) changes of calcium and total Alkaline Phosphatase (tAP) plasma levels during treatment with NER and (2) tolerability of 100 mg NER monthly administration for 12 months. These data suggest that NER, if administered at these doses and timing, might allow at least for one year sustained BMD increases in patients. NER has been highly tolerated in this study. The almost complete absence of adverse effects has prompted us to reduce the time of infusions at the end of the study. In conclusion, this study provides the first data on the efficacy and safety of NER in patients with MM-induced bone damage. These initial data encourage wider phase III trials to clearly assess its efficacy in preventing skeletal-related events and its possible anti-neoplastic properties.
Authors:
Gianfranco Pittari; Daniele Costi; Marco Raballo; Linda Maulucci; Maria Cristina Baroni; Marcellina Mangoni
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Publication Detail:
Type:  Comparative Study; Evaluation Studies; Journal Article    
Journal Detail:
Title:  Acta bio-medica : Atenei Parmensis     Volume:  77     ISSN:  0392-4203     ISO Abbreviation:  -     Publication Date:  2006 Aug 
Date Detail:
Created Date:  2006-12-18     Completed Date:  2007-01-05     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101295064     Medline TA:  Acta Biomed     Country:  Italy    
Other Details:
Languages:  eng     Pagination:  81-4     Citation Subset:  IM    
Affiliation:
Hematology and Bone Trasplantation Unit, University of Parma, Italy.
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MeSH Terms
Descriptor/Qualifier:
Aged
Alkaline Phosphatase / blood
Bone Density
Bone Resorption / blood,  drug therapy*
Calcium / blood
Diphosphonates / administration & dosage*
Female
Hip
Humans
Infusions, Intravenous
Lumbar Vertebrae
Male
Middle Aged
Multiple Myeloma / complications*,  mortality
Osteoclasts / physiology
Osteolysis / drug therapy
Safety
Time Factors
Treatment Outcome
Chemical
Reg. No./Substance:
0/Diphosphonates; 7440-70-2/Calcium; 79778-41-9/6-amino-1-hydroxyhexane-1,1-diphosphonate; EC 3.1.3.1/Alkaline Phosphatase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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