Document Detail


Intracoronary endothelin-1 increases coronary retrograde pressure by constricting arterioles.
MedLine Citation:
PMID:  2097065     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
STUDY OBJECTIVE: The aim was to determine the site of coronary vasoconstriction induced by endothelin, by investigating the response in terms of retrograde pressure and reactive hyperaemia. EXPERIMENTAL MATERIAL: Twelve anaesthetised mongrel dogs, 12-14 kg, were used for the studies. DESIGN: The left anterior descending coronary artery was cannulated and perfused with blood through an extracorporeal bypass. The effects of intracoronary endothelin-1 (1-500 pmol) on coronary blood flow, coronary flow reserve (the peak reactive flow and the repayment after 15 s coronary occlusion), and retrograde coronary pressure during coronary occlusion were studied (n = 7). The retrograde coronary flow was collected from the bypass at each dose (n = 5). MEASUREMENTS AND MAIN RESULTS: At doses of greater than 20 pmol the coronary flow decreased dose dependently and reached almost zero flow at 500 pmol. The coronary flow reserve also decreased; however, the retrograde pressure was raised dose dependently at doses of greater than 10 pmol. At a dose of 500 pmol, the retrograde pressure was increased to 61 mm Hg [82(SEM 12)% of the coronary perfusion pressure]. Retrograde flow remained unchanged throughout the experiment. CONCLUSIONS: The endothelin-1 induced rise in retrograde pressure is in accordance with a dose dependent reduction in coronary flow reserve, and collateral flow was not augmented by endothelin. It is concluded that the effect of endothelin-1 on coronary circulation in situ was mainly due to the constriction of small resistant vessels.
Authors:
K Fukuda; S Hori; M Kusuhara; T Satoh; S Kyotani; S Inoue; H Ohno; K Yamaguchi; S Handa; Y Nakamura
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Cardiovascular research     Volume:  24     ISSN:  0008-6363     ISO Abbreviation:  Cardiovasc. Res.     Publication Date:  1990 Dec 
Date Detail:
Created Date:  1991-07-22     Completed Date:  1991-07-22     Revised Date:  2010-03-24    
Medline Journal Info:
Nlm Unique ID:  0077427     Medline TA:  Cardiovasc Res     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  987-92     Citation Subset:  IM    
Affiliation:
Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Pressure / drug effects
Coronary Circulation / drug effects
Coronary Vessels / drug effects*
Dogs
Dose-Response Relationship, Drug
Endothelins / pharmacology*
Vasoconstriction / drug effects*
Chemical
Reg. No./Substance:
0/Endothelins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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