Document Detail


Interleukin-6 is a novel factor mediating glucose homeostasis during skeletal muscle contraction.
MedLine Citation:
PMID:  15220185     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The mechanisms that mediate the tightly controlled production and clearance of glucose during muscular work are unclear, and it has been suggested that an unidentified "work factor" exists that influences the contraction-induced increase in endogenous glucose production (EGP). The cytokine interleukin (IL)-6 is released from skeletal muscle during contraction. Here we show that IL-6 contributes to the contraction-induced increase in EGP. Six men performed 2 h of bicycle exercise on three separate occasions, at a relatively high intensity (HI) or at a low intensity with (LO + IL-6) or without (LO) an infusion of recombinant human IL-6 that matched the circulating concentration of IL-6 seen in HI exercise. The stable isotope 6,6 (2)H(2) glucose was infused to calculate EGP (rate of glucose appearance [R(a)]), whole-body glucose disposal (rate of glucose disappearance [R(d)]), and metabolic clearance rate (MCR) of glucose. Glucose R(a), R(d), and MCR were higher (P < 0.05) at HI than at LO. Throughout exercise at LO + IL-6, glucose R(a) and R(d) were higher (P < 0.05) than LO, even though the exercise intensity was identical. In addition, MCR was higher (P < 0.05) at LO + IL-6 than at LO at 90 min. Insulin, glucagon, epinephrine, norepinephrine, cortisol, and growth hormone were identical when comparing LO + IL-6 with LO. These data suggest that IL-6 influences glucose homeostasis during exercise. Our results provide potential new insights into factors that mediate glucose production and disposal and implicates IL-6 in the so-called "work factor."
Authors:
Mark A Febbraio; Natalie Hiscock; Massimo Sacchetti; Christian P Fischer; Bente K Pedersen
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Publication Detail:
Type:  Clinical Trial; Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Diabetes     Volume:  53     ISSN:  0012-1797     ISO Abbreviation:  Diabetes     Publication Date:  2004 Jul 
Date Detail:
Created Date:  2004-06-28     Completed Date:  2004-09-16     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0372763     Medline TA:  Diabetes     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1643-8     Citation Subset:  AIM; IM    
Affiliation:
Skeletal Muscle Research Laboratory, Center for Nutrition, Metabolism and Endocrinology, RMIT University, P.O. Box 71, Bundoora 3083, Victoria, Australia. mark.febbraio@rmit.edu.au
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MeSH Terms
Descriptor/Qualifier:
Adult
Bicycling
Exercise / physiology
Fatty Acids, Nonesterified / blood
Glucose / biosynthesis,  metabolism*
Homeostasis / physiology*
Hormones / blood
Humans
Interleukin-6 / pharmacology,  physiology*
Lactic Acid / blood
Male
Metabolic Clearance Rate
Muscle Contraction / physiology*
Muscle, Skeletal / physiology*
Recombinant Proteins / pharmacology
Chemical
Reg. No./Substance:
0/Fatty Acids, Nonesterified; 0/Hormones; 0/Interleukin-6; 0/Recombinant Proteins; 50-21-5/Lactic Acid; 50-99-7/Glucose

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