Document Detail


Interleukin-27 as a negative regulator of human neutrophil function.
MedLine Citation:
PMID:  20883313     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Interleukin-27 (IL-27) is a novel cytokine of the IL-6/12 family with a broad range of immune regulation properties, which has been considered as a potential therapeutic agent for immune diseases and cancers. However, little is known about the effect of IL-27 on human neutrophils before its clinical administration. In this study, we investigated the effects of IL-27 on human neutrophil functions including adhesion, reactive oxygen species (ROS)/cytotoxic granule components production, inflammatory cytokines production, major histocompatibility complex (MHC) molecules expression and neutrophils' survival. We showed that IL-27 receptor complex, WSX-1/TCCR and gp130, is constitutively expressed on human neutrophils. In vitro, IL-27 suppressed neutrophil adhesion in response to fMLP, which might depend on the down-regulation of Mac-1. IL-27 also suppressed lipopolysaccharide-induced ROS production and attenuated cytotoxic granule components production in the cytoplasm of human neutrophils. In addition, IL-27 enhanced the production of IL-1β but not TNF-α from neutrophils. However, IL-27 failed to regulate the expression of MHC molecules and the survival of human neutrophils. In conclusion, our data demonstrate that IL-27 mainly down-modulates human neutrophil function, which might extend our understanding of the role of IL-27 in the innate immune response.
Authors:
J P Li; H Wu; W Xing; S G Yang; S H Lu; W T Du; J X Yu; F Chen; L Zhang; Z C Han
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Scandinavian journal of immunology     Volume:  72     ISSN:  1365-3083     ISO Abbreviation:  Scand. J. Immunol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-04     Completed Date:  2010-11-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0323767     Medline TA:  Scand J Immunol     Country:  England    
Other Details:
Languages:  eng     Pagination:  284-92     Citation Subset:  IM    
Copyright Information:
© 2010 The Authors. Scandinavian Journal of Immunology © 2010 Blackwell Publishing Ltd.
Affiliation:
State Key Laboratory of Experimental Hematology, Institute of Hematology and Hospital of Blood Diseases, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
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MeSH Terms
Descriptor/Qualifier:
Cell Adhesion / drug effects
Cell Nucleus / drug effects,  ultrastructure
Cell Survival / drug effects
Cells, Cultured
Cytokine Receptor gp130 / genetics,  metabolism
Cytokines / metabolism*
Cytoplasmic Granules / drug effects,  metabolism
Flow Cytometry
Gene Expression Regulation / drug effects
Humans
Inflammation Mediators / metabolism
Interleukin-1beta / metabolism
Interleukins / pharmacology*
Lymphocyte Function-Associated Antigen-1 / genetics,  metabolism
Macrophage-1 Antigen / genetics,  metabolism
Microscopy, Electron, Transmission
Mitochondria / drug effects,  ultrastructure
Neutrophils / cytology,  drug effects*,  metabolism
Reactive Oxygen Species / metabolism*
Receptors, Interleukin / genetics,  metabolism
Reverse Transcriptase Polymerase Chain Reaction
Tumor Necrosis Factor-alpha / metabolism
Chemical
Reg. No./Substance:
0/C19orf10 protein, human; 0/Cytokines; 0/IL27RA protein, human; 0/IL6ST protein, human; 0/Inflammation Mediators; 0/Interleukin-1beta; 0/Interleukins; 0/Lymphocyte Function-Associated Antigen-1; 0/Macrophage-1 Antigen; 0/Reactive Oxygen Species; 0/Receptors, Interleukin; 0/Tumor Necrosis Factor-alpha; 133483-10-0/Cytokine Receptor gp130

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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