Document Detail


Interleukin 21 up-regulates perforin-mediated cytotoxic activity of human intra-epithelial lymphocytes.
MedLine Citation:
PMID:  19489126     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Human intra-epithelial lymphocytes (IELs) are predominantly T-cell receptor-alphabeta(+) (TCR-alphabeta(+)) CD8(+) CD45RO(+) memory T cells located between intestinal epithelial cells. They respond to a greater extent to stimulation with interleukin (IL)-15 than to CD3/TCR triggering, suggesting that they react to the cytokine milieu in their local environment rather than to cognate antigen. A newly described member of the gammac cytokine family, IL-21, has potent antitumor effects. As IELs resemble lymphocytes infiltrating neoplastic lesions, their response to IL-21 may be relevant in vivo. Here, IL-21 was shown to increase perforin-mediated cytotoxicity and serine esterase release by IELs. This IL-21-mediated up-regulation occurred without changes in IEL survival or cell division. Interestingly, the effects of IL-21 occurred without increased phosphorylation of signal transducer and activator of transcription (STAT)1, STAT3, STAT4, STAT5, extracellular signal-regulated kinase (ERK), or p38. IL-21 had no effect on Fas ligand (FL)- or tumour necrosis factor-alpha (TNF-alpha)-mediated cytotoxicity, but it down-regulated IL-15-stimulated expression of CD25 and CD94, indicating that it has both positive and negative actions. This functional profile is unique to human IELs, emphasizing that they are a distinct compartment of lymphocytes and that IL-21 may promote their role in tumour immunosurveillance.
Authors:
Ellen C Ebert
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Immunology     Volume:  127     ISSN:  1365-2567     ISO Abbreviation:  Immunology     Publication Date:  2009 Jun 
Date Detail:
Created Date:  2009-06-01     Completed Date:  2009-07-31     Revised Date:  2010-09-24    
Medline Journal Info:
Nlm Unique ID:  0374672     Medline TA:  Immunology     Country:  England    
Other Details:
Languages:  eng     Pagination:  206-15     Citation Subset:  IM    
Affiliation:
UMDNJ-Robert Wood Johnson Medical School, New Brunswick, NJ, USA. ebertec@umdnj.edu
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MeSH Terms
Descriptor/Qualifier:
Cell Proliferation
Cell Survival / immunology
Cells, Cultured
Cytoplasmic Granules / immunology
Cytotoxicity, Immunologic / immunology
Epithelial Cells / immunology
Fas Ligand Protein / immunology
Humans
Immunity, Mucosal
Interleukin-15 / immunology
Interleukins / immunology*
Intestinal Mucosa / immunology*
Jejunum / immunology
Perforin / immunology*
Phosphorylation / immunology
STAT Transcription Factors / immunology
Tumor Necrosis Factor-alpha / immunology
Up-Regulation / immunology
Chemical
Reg. No./Substance:
0/Fas Ligand Protein; 0/Interleukin-15; 0/Interleukins; 0/STAT Transcription Factors; 0/Tumor Necrosis Factor-alpha; 0/interleukin-21; 126465-35-8/Perforin
Comments/Corrections

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