Document Detail


Interleukin 2 receptor-targeted therapy--rationale and applications in organ transplantation.
MedLine Citation:
PMID:  2462756     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Despite major advances in immunopharmacology for use in clinical organ transplantation, graft rejection and drug-induced side effects remain the major problems with currently available immunosuppressive modalities. Recent advances in hybridoma technology have produced relatively effective and reproducible biological immunosuppression with monoclonal antibodies; indeed, OKT3 and anti-T12 mAbs have been employed with considerable success as adjuncts to chemical suppression in treating rejection. Nonetheless, the use of such antibodies broadly reactive to differentiation antigens on T lymphocytes does not solve the problems of side effects caused by general immunosuppression. An ideal therapeutic agent should target only lymphocytes that participate in rejection of foreign tissue without affecting physiological host immune surveillance and normal defense mechanisms. Theoretically, this goal could be achieved by "antigenic suicide," or by using the appropriate antiidiotypic antibodies or mAbs against the antigen combining site of T cell receptors. However, because of the intense polymorphism of transplantation antigens and the vast genetic repertoire encoding for the T cell antigen receptor, success of such forms of specific immunosuppression, at least at this time, is highly improbable.
Authors:
J W Kupiec-Weglinski; T Diamantstein; N L Tilney
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Transplantation     Volume:  46     ISSN:  0041-1337     ISO Abbreviation:  Transplantation     Publication Date:  1988 Dec 
Date Detail:
Created Date:  1989-02-06     Completed Date:  1989-02-06     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0132144     Medline TA:  Transplantation     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  785-92     Citation Subset:  IM    
Affiliation:
Surgical Research Laboratory, Harvard Medical School, Boston, MA 02115.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antibodies, Monoclonal / therapeutic use*
Antigens, Differentiation, T-Lymphocyte / immunology
Epitopes / immunology
Graft Rejection
Humans
Immunosuppression / methods*
Kidney Transplantation
Lymphocyte Activation
Macaca
Mice
Papio
Receptors, Interleukin-2 / immunology*
Transplantation Immunology*
Grant Support
ID/Acronym/Agency:
R01 AI19071/AI/NIAID NIH HHS; R01 AI23847/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Antigens, Differentiation, T-Lymphocyte; 0/Epitopes; 0/Receptors, Interleukin-2

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