Document Detail


Interleukin-13 protects mouse intestine from ischemia and reperfusion injury through regulation of innate and adaptive immunity.
MedLine Citation:
PMID:  21311412     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Ischemia-reperfusion (I/R) injury is a major factor leading to intestinal dysfunction or graft loss after intestinal surgery or transplantation. This study investigated the cytoprotective effects and putative mechanisms of interleukin (IL)-13 after intestinal I/R injury in the mouse.
METHODS: Mouse warm intestinal I/R injury induced by clamping the superior mesenteric artery for 100 min with tissue analysis at 4 and 24 hr after reperfusion. Treated animals received intravenous recombinant murine IL-13 (rIL-13) and anti-IL-13 antibody, whereas controls received saline.
RESULTS: rIL-13 administration markedly prolonged animal survival (100% vs. 50% in saline controls) and resulted in near normal histopathological architecture. rIL-13 treatment also significantly decreased myeloperoxidase activity. Mice conditioned with rIL-13 had a markedly depressed Toll-like receptor-4 expression and increased the expression of Stat6, antioxidant hemeoxygenase-1, and antiapoptotic A20, Bcl-2/Bcl-xl, compared with that of controls. Unlike in controls, the expression of mRNA coding for IL-2/interferon-γ, and interferon-γ-inducible protein (IP)-10/monocyte chemotactic protein-1 remained depressed, whereas that of IL-13/IL-4 reciprocally increased in the mice treated with rIL-13. Administration of anti-IL13 antibody alone or in combination with rIL-13 resulted in outcomes similar to that seen in controls.
CONCLUSIONS: This study demonstrates for the first time that IL-13 plays a protective role in intestinal warm I/R injury and a critical role in the regulation of Stat6 and Toll-like receptor-4 signaling. The administration of IL-13 exerts cytoprotective effects in this model by regulating innate and adaptive immunity while the removal of IL-13 using antibody therapy abrogates this effect.
Authors:
Douglas G Farmer; Bibo Ke; Xiu-Da Shen; Fady M Kaldas; Feng Gao; Melissa J Watson; Ronald W Busuttil; Jerzy W Kupiec-Weglinski
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Transplantation     Volume:  91     ISSN:  1534-6080     ISO Abbreviation:  Transplantation     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-03-25     Completed Date:  2011-06-01     Revised Date:  2012-01-18    
Medline Journal Info:
Nlm Unique ID:  0132144     Medline TA:  Transplantation     Country:  United States    
Other Details:
Languages:  eng     Pagination:  737-43     Citation Subset:  IM    
Affiliation:
Division of Liver and Pancreas Transplantation, Department of Surgery, The Dumont-UCLA Transplant Center, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA. dgfarmer@mednet.ucla.edu
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MeSH Terms
Descriptor/Qualifier:
Adaptive Immunity*
Animals
Cytoprotection
Immunity, Innate*
Interleukin-13 / blood,  pharmacology*
Intestines / blood supply*
Male
Mice
Mice, Inbred C57BL
Peroxidase / metabolism
Recombinant Proteins / pharmacology
Reperfusion Injury / prevention & control*
STAT6 Transcription Factor / physiology
Toll-Like Receptor 4 / physiology
Grant Support
ID/Acronym/Agency:
R01 DK062357-09/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Interleukin-13; 0/Recombinant Proteins; 0/STAT6 Transcription Factor; 0/Stat6 protein, mouse; 0/Tlr4 protein, mouse; 0/Toll-Like Receptor 4; EC 1.11.1.7/Peroxidase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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