Document Detail


Interleukin-1 resets glucose homeostasis at central and peripheral levels: relevance for immunoregulation.
MedLine Citation:
PMID:  20134186     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We briefly discuss here evidence showing that the capacity of IL-1beta to mediate adjustments of glucose homeostasis can be added to the already well-known pleiotropic effects of this cytokine. Such adjustments, which are necessary for satisfying the high energetic demands of immune/inflammatory responses, can be mediated by effects of endogenous IL-1 exerted at peripheral and brain levels in a concerted action with other cytokines and neuroendocrine mechanisms.
Authors:
Hugo O Besedovsky; Adriana Del Rey
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2010-02-04
Journal Detail:
Title:  Neuroimmunomodulation     Volume:  17     ISSN:  1423-0216     ISO Abbreviation:  Neuroimmunomodulation     Publication Date:  2010  
Date Detail:
Created Date:  2010-02-05     Completed Date:  2010-06-14     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9422763     Medline TA:  Neuroimmunomodulation     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  139-41     Citation Subset:  IM    
Copyright Information:
Copyright 2010 S. Karger AG, Basel.
Affiliation:
Department of Immunophysiology, Institute of Physiology and Pathophysiology, Medical Faculty, Marburg, Germany. besedovs@mailer.uni-marburg.de
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MeSH Terms
Descriptor/Qualifier:
Animals
Energy Metabolism / immunology*
Glucose / metabolism*
Glucose Transporter Type 4 / metabolism
Homeostasis / immunology
Humans
Immune System / physiology
Insulin / metabolism,  secretion
Interleukin-1beta / metabolism*
Neuroimmunomodulation / physiology*
Chemical
Reg. No./Substance:
0/Glucose Transporter Type 4; 0/Interleukin-1beta; 11061-68-0/Insulin; 50-99-7/Glucose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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