Document Detail


Insulin delays the progression of Drosophila cells through G2/M by activating the dTOR/dRaptor complex.
MedLine Citation:
PMID:  17183368     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In Drosophila and mammals, insulin signalling can increase growth, progression through G1/S, cell size and tissue size. Here, we analyse the way insulin affects cell size and cell-cycle progression in two haemocyte-derived Drosophila cell lines. Surprisingly, we find that although insulin increases cell size, it slows the rate at which these cells increase in number. By using BrdU pulse-chase to label S-phase cells and follow their progression through the cell cycle, we show that insulin delays progression through G2/M, thereby slowing cell division. The ability of insulin to slow progression through G2/M is independent of its ability to stimulate progression through G1/S, so is not a consequence of feedback by the cell-cycle machinery to maintain cell-cycle length. Insulin's effects on progression through G2/M are mediated by dTOR/dRaptor signalling. Partially inhibiting dTOR/dRaptor signalling by dsRNAi or mild rapamycin treatment can increase cell number in cultured haemocytes and the Drosophila wing, respectively. Thus, insulin signalling can influence cell number depending on a balance between its ability to accelerate progression through G1/S and delay progression through G2/M.
Authors:
Mary Y W Wu; Megan Cully; Ditte Andersen; Sally J Leevers
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2006-12-21
Journal Detail:
Title:  The EMBO journal     Volume:  26     ISSN:  0261-4189     ISO Abbreviation:  EMBO J.     Publication Date:  2007 Jan 
Date Detail:
Created Date:  2007-01-24     Completed Date:  2007-03-27     Revised Date:  2010-02-01    
Medline Journal Info:
Nlm Unique ID:  8208664     Medline TA:  EMBO J     Country:  England    
Other Details:
Languages:  eng     Pagination:  371-9     Citation Subset:  IM    
Affiliation:
Growth Regulation Laboratory, Cancer Research UK London Research Institute, London, UK.
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MeSH Terms
Descriptor/Qualifier:
1-Phosphatidylinositol 3-Kinase / metabolism*
Animals
Cell Division / drug effects*
Cell Proliferation / drug effects
Cells, Cultured
Drosophila
Drosophila Proteins / metabolism*
G2 Phase / drug effects*
Insulin / pharmacology*
Intracellular Signaling Peptides and Proteins / metabolism*
Male
Multiprotein Complexes / metabolism
RNA Interference
S Phase / drug effects
Signal Transduction
Wing / cytology,  drug effects
Chemical
Reg. No./Substance:
0/Drosophila Proteins; 0/Intracellular Signaling Peptides and Proteins; 0/Multiprotein Complexes; 0/Raptor protein, Drosophila; 11061-68-0/Insulin; EC 2.7.1.-/target of rapamycin protein, Drosophila; EC 2.7.1.137/1-Phosphatidylinositol 3-Kinase
Comments/Corrections

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