Document Detail


Instabilities of metabolic regulations in aging.
MedLine Citation:
PMID:  1450606     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
1) That non-invasive NMR and optical methods can a) quantify the work stress on mitochondria for ATP production, and b) indicate the tissue O2 tension in the capillary bed that is responsible for the rate of radical generation. 2) That free radical damage to mitochondrial function can be quantified by reciprocal plots of inverse slope giving the extrapolated Vm of mitochondria. 3) That a particular genetically deficient individual requiring high dosages of menadione has survived over 9 years. 4) That mitochondrial deficiency leads to an exercise hyperoxia.
Authors:
B Chance; W W Bank; C Zhang
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  EXS     Volume:  62     ISSN:  1023-294X     ISO Abbreviation:  EXS     Publication Date:  1992  
Date Detail:
Created Date:  1993-01-06     Completed Date:  1993-01-06     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  9204529     Medline TA:  EXS     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  58-63     Citation Subset:  IM; S    
Affiliation:
Department of Biochemistry and Biophysics, Johnson Foundation, University of Pennsylvania, Philadelphia 19104.
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MeSH Terms
Descriptor/Qualifier:
Adenosine Diphosphate / metabolism
Adenosine Triphosphate / metabolism
Aging / metabolism*
Animals
Free Radicals / metabolism
Homeostasis
Humans
Magnetic Resonance Spectroscopy / methods
Mitochondria / metabolism*
Models, Biological
Muscles / metabolism
Physical Exertion
Grant Support
ID/Acronym/Agency:
AA07186/AA/NIAAA NIH HHS; HL18708/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Free Radicals; 56-65-5/Adenosine Triphosphate; 58-64-0/Adenosine Diphosphate

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