Document Detail


Innate immune activation during Salmonella infection initiates extramedullary erythropoiesis and splenomegaly.
MedLine Citation:
PMID:  20952675     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Systemic Salmonella infection commonly induces prolonged splenomegaly in murine or human hosts. Although this increase in splenic cellularity is often assumed to be due to the recruitment and expansion of leukocytes, the actual cause of splenomegaly remains unclear. We monitored spleen cell populations during Salmonella infection and found that the most prominent increase is found in the erythroid compartment. At the peak of infection, the majority of spleen cells are immature CD71(-)Ter119(+) reticulocytes, indicating that massive erythropoiesis occurs in response to Salmonella infection. Indeed, this increase in RBC precursors corresponded with marked elevation of serum erythropoietin (EPO). Furthermore, the increase in RBC precursors and EPO production required innate immune signaling mediated by Myd88/TRIF. Neutralization of EPO substantially reduced the immature RBC population in the spleen and allowed a modest increase in host control of infection. These data indicate that early innate immunity to Salmonella initiates marked splenic erythropoiesis and may hinder bacterial clearance.
Authors:
Amy Jackson; Minelva R Nanton; Hope O'Donnell; Adovi D Akue; Stephen J McSorley
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-10-15
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  185     ISSN:  1550-6606     ISO Abbreviation:  J. Immunol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-11-04     Completed Date:  2010-12-02     Revised Date:  2013-05-14    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  6198-204     Citation Subset:  AIM; IM    
Affiliation:
Center for Infectious Diseases and Microbiology Translational Research, Division of Gastroenterology, Hepatology and Nutrition, Department of Medicine, University of Minnesota Medical School, Minneapolis, MN 55455, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Erythropoiesis / immunology*
Erythropoietin / blood
Flow Cytometry
Immunity, Innate / immunology*
Immunoassay
Mice
Mice, Inbred C57BL
Mice, Transgenic
Reticulocytes / immunology
Salmonella Infections / blood,  immunology*,  physiopathology
Splenomegaly / immunology*,  microbiology,  parasitology
Grant Support
ID/Acronym/Agency:
AI055743/AI/NIAID NIH HHS; AI076278/AI/NIAID NIH HHS; AI56172/AI/NIAID NIH HHS; P01 AI056172-06A2/AI/NIAID NIH HHS; R01 AI055743/AI/NIAID NIH HHS; R01 AI055743-08/AI/NIAID NIH HHS; R01 AI076278/AI/NIAID NIH HHS; R01 AI076278-01A1/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
11096-26-7/Erythropoietin
Comments/Corrections

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