Document Detail


Inhibition of poly(ADP-ribose) polymerase prevents allergen-induced asthma-like reaction in sensitized Guinea pigs.
MedLine Citation:
PMID:  15254147     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Poly(ADP-ribose) polymerase (PARP) plays an important role in tissue injury in conditions associated with oxidative stress and inflammation. Because asthma is a chronic inflammatory disorder of the airways, we designed the present experimental study to evaluate the effects of PARP inhibition on allergen-induced asthma-like reaction in ovalbumin-sensitized guinea pigs. Cough and dyspnea in response to ovalbumin aerosol were absent in naive guinea pigs, whereas they became severe in the sensitized animals. In the latter ones, ovalbumin aerosol also induced a rapid increase in PARP activity, bronchiolar constriction, pulmonary air space inflation, mast cell degranulation, poly(ADP-ribose) and nitrotyrosine immunostaining, myeloperoxidase activity, and malondialdehyde in lung tissue, as well as a rise in the amounts of nitrites and tumor necrosis factor-alpha in bronchoalveolar lavage fluid. Pretreatment with the PARP inhibitors 3-aminobenzamide (10 mg/kg b.wt.) or 5-aminoisoquinolinone (0.5 mg/kg b.wt.) given i.p. 3 h before ovalbumin challenge significantly reduced the severity of cough and the occurrence of dyspnea and delayed the onset of respiratory abnormalities. Both PARP inhibitors were also able to prevent the above morphological and biochemical changes of lung tissue or bronchoalveolar lavage fluid induced by ovalbumin challenge. Conversely, p-aminobenzoic acid, the inactive analog of 3-aminobenzamide, had no effects.
Authors:
Ylenia Suzuki; Emanuela Masini; Cosimo Mazzocca; Salvatore Cuzzocrea; Anna Ciampa; Hisanori Suzuki; Daniele Bani
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2004-07-13
Journal Detail:
Title:  The Journal of pharmacology and experimental therapeutics     Volume:  311     ISSN:  0022-3565     ISO Abbreviation:  J. Pharmacol. Exp. Ther.     Publication Date:  2004 Dec 
Date Detail:
Created Date:  2004-11-23     Completed Date:  2005-01-13     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0376362     Medline TA:  J Pharmacol Exp Ther     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1241-8     Citation Subset:  IM    
Affiliation:
Department of Anatomy, Histology, and Forensic Medicine, Section of Histology, University of Florence, Viale G. Pieraccini, 6, I-50139 Florence, Italy.
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MeSH Terms
Descriptor/Qualifier:
Allergens*
Animals
Anti-Asthmatic Agents*
Asthma / physiopathology,  prevention & control*
Benzamides / pharmacology
Bronchoalveolar Lavage Fluid / chemistry
Enzyme Inhibitors / pharmacology*
Guinea Pigs
Immunohistochemistry
Isoquinolines / pharmacology
Lung / pathology
Male
Malondialdehyde / metabolism
Mast Cells / drug effects,  metabolism
Nitric Oxide / metabolism
Ovalbumin / immunology
Peroxidase / metabolism
Poly(ADP-ribose) Polymerases / antagonists & inhibitors*
Respiratory Mechanics / drug effects,  physiology
Tumor Necrosis Factor-alpha / metabolism
Tyrosine / analogs & derivatives*,  pharmacology
Chemical
Reg. No./Substance:
0/5-aminoisoquinolinone; 0/Allergens; 0/Anti-Asthmatic Agents; 0/Benzamides; 0/Enzyme Inhibitors; 0/Isoquinolines; 0/Tumor Necrosis Factor-alpha; 10102-43-9/Nitric Oxide; 3544-24-9/3-aminobenzamide; 3604-79-3/3-nitrotyrosine; 542-78-9/Malondialdehyde; 55520-40-6/Tyrosine; 9006-59-1/Ovalbumin; EC 1.11.1.7/Peroxidase; EC 2.4.2.30/Poly(ADP-ribose) Polymerases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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