Document Detail


Inhibition of cerebral ischemia/reperfusion-induced injury by adenovirus expressed C-terminal amino acids of GluR6.
MedLine Citation:
PMID:  19747468     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
GluR6 kainate receptor subunit is largely expressed in hippocampus of brain regions and plays an important role in brain ischemia/reperfusion-mediated neuronal cell death. Our previous researches have shown that cerebral ischemia/reperfusion could facilitate the assembly of GluR6 and postsynaptic density protein 95(PSD95) as well as mixed lineage kinase 3(MLK3) and further induce the activation of c-Jun NH2-terminal kinase 3(JNK3), leading to neuronal death of hippocampal CA1. Here, we show that over-expression of C-terminal amino acids of GluR6 can interrupt the combination of GluR6 with PSD95, inhibit the assembly of GluR6.PSD-95.MLK3 signaling module, suppress the activation of JNK3 and the downstream signaling pathway. Thus, our results imply that over-expression of C-terminal amino acids of GluR6 induce neuroprotection against ischaemic brain injury in rat hippocampal CA1 region via suppressing proapoptosis signaling pathways, which can be an experimental foundation for gene therapy of stroke.
Authors:
Ting Li; Hong-Min Yu; Ya-Feng Sun; Yuan-Jian Song; Guang-Yi Zhang; Dong-Sheng Pei
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-09-09
Journal Detail:
Title:  Brain research     Volume:  1300     ISSN:  1872-6240     ISO Abbreviation:  Brain Res.     Publication Date:  2009 Dec 
Date Detail:
Created Date:  2009-10-27     Completed Date:  2010-01-05     Revised Date:  2010-01-13    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  169-76     Citation Subset:  IM    
Affiliation:
Research Center for Biochemistry and Molecular Biology, Xuzhou Medical College, Xuzhou 221002, China.
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MeSH Terms
Descriptor/Qualifier:
Adenoviridae / metabolism
Analysis of Variance
Animals
Blotting, Western
Brain Ischemia / metabolism,  pathology,  therapy*
CA1 Region, Hippocampal / metabolism,  pathology
Cell Death / physiology
Cell Fractionation
Cell Survival / genetics
Cytoprotection
Gene Transfer Techniques
Genetic Vectors
Intracellular Signaling Peptides and Proteins / metabolism
MAP Kinase Kinase Kinases / metabolism
Male
Membrane Proteins / metabolism
Mitogen-Activated Protein Kinase 10 / metabolism
Neurons / metabolism
Phosphorylation
Rats
Rats, Sprague-Dawley
Receptors, Kainic Acid / metabolism*
Reperfusion Injury / metabolism,  pathology,  therapy*
Signal Transduction / physiology
Staining and Labeling
Subcellular Fractions / metabolism
Chemical
Reg. No./Substance:
0/Dlgh4 protein, rat; 0/Gluk2 kainate receptor; 0/Intracellular Signaling Peptides and Proteins; 0/Membrane Proteins; 0/Receptors, Kainic Acid; EC 2.7.1.-/Mitogen-Activated Protein Kinase 10; EC 2.7.11.25/MAP Kinase Kinase Kinases; EC 2.7.11.25/mitogen-activated protein kinase kinase kinase 11

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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