Document Detail


Inhibition of amyloid-beta-induced neurotoxicity and apoptosis by moderate ethanol preconditioning.
MedLine Citation:
PMID:  15486488     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Consumers of moderate amounts of ethanol have a lower risk of Alzheimer's dementia than do abstainers. In Alzheimer's disease the brain contains many extracellular plaques composed of amyloid-beta (Abeta), a neurotoxic protein linked to pathogenesis of the disease. Here we report that moderate ethanol preconditioning (20-30 mM for 6 days) of organotypic hippocampal-entorhinal slice cultures prevents Abeta-induced neurotoxicity and apoptosis as measured by media lactate dehydrogenase levels and staining with propidium iodide and Hoechst 33342. With Abeta, as with our previous studies of the neurotoxic HIV-1 protein gp120, moderate ethanol preconditioning may interfere with various glial-mediated neurotoxic responses in the slices to Abeta. In addition, we found that moderate ethanol preconditioning causes an almost 3-fold increase in brain levels of heat shock protein 70 (hsp70), a protective molecular chaperone. Our results suggest possible molecular mechanisms underlying the protective effect of moderate drinking against Alzheimer's dementia.
Authors:
Abdelhak Belmadani; Sripathirathan Kumar; Matthew Schipma; Michael A Collins; Edward J Neafsey
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Publication Detail:
Type:  Comparative Study; In Vitro; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Neuroreport     Volume:  15     ISSN:  0959-4965     ISO Abbreviation:  Neuroreport     Publication Date:  2004 Sep 
Date Detail:
Created Date:  2004-10-15     Completed Date:  2005-01-13     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9100935     Medline TA:  Neuroreport     Country:  England    
Other Details:
Languages:  eng     Pagination:  2093-6     Citation Subset:  IM    
Affiliation:
Division of Biochemistry, Department of Cell Biology, Neurobiology and Anatomy, Loyola University Medical Center, 2160 S. First Avenue, Maywood, IL 60153, USA.
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MeSH Terms
Descriptor/Qualifier:
Amyloid beta-Protein / toxicity*
Animals
Animals, Newborn
Apoptosis / drug effects*
Benzimidazoles / diagnostic use
Central Nervous System Depressants / pharmacology*,  therapeutic use
Dose-Response Relationship, Drug
Drug Interactions
Enzyme-Linked Immunosorbent Assay / methods
Ethanol / pharmacology*,  therapeutic use
HSP70 Heat-Shock Proteins / metabolism
Hippocampus / drug effects*
Hydro-Lyases / metabolism
Nerve Degeneration / etiology,  prevention & control*
Organ Culture Techniques
Propidium / diagnostic use
Rats
Rats, Sprague-Dawley
Grant Support
ID/Acronym/Agency:
AA11723/AA/NIAAA NIH HHS
Chemical
Reg. No./Substance:
0/Amyloid beta-Protein; 0/Benzimidazoles; 0/Central Nervous System Depressants; 0/HSP70 Heat-Shock Proteins; 23491-52-3/HOE 33342; 36015-30-2/Propidium; 64-17-5/Ethanol; EC 4.2.1.-/Hydro-Lyases; EC 4.2.1.54/lactate dehydratase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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