Document Detail


Influence of proton availability on intracapillary CO2-HCO3(-)-H+ reactions in isolated rat lungs.
MedLine Citation:
PMID:  1321108     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Transcapillary CO2 exchange entails a transient perfusate CO2-HCO3(-)-H+ disequilibrium, leading to net loading or unloading of blood HCO3-. Perfusate reequilibration may or may not reach completion during the time of capillary transit, depending on the rate of intracapillary CO2-HCO3(-)-H+ reactions. Failure to reestablish equilibrium within the "open" capillary system leads to continued reequilibration in the "closed" postcapillary vasculature with resultant shifts in postcapillary perfusate PCO2, pH, and [HCO3-]. In the present study, we determined the effects of perfusate nonbicarbonate buffer capacity (beta) on intracapillary CO2-HCO3(-)-H+ reactions in isolated saline-perfused rat lungs. Effects of beta on the rate of transcapillary CO2 excretion (VCO2) and the magnitude of the postcapillary perfusate pH disequilibrium were measured as a function of luminal vascular carbonic anhydrase (CA) activity. The data indicate that beta markedly influenced the kinetics and dynamics of intravascular CO2-HCO3(-)-H+ reactions. beta affected VCO2 and the relative enhancement of VCO2 by luminal vascular CA. The data emphasize the inadequacies of using traditional "equilibrium" models of the CO2-HCO3(-)-H+ system to investigate capillary CO2 transport and exchange, even in organs (e.g., lungs) that contain significant luminal vascular CA activity.
Authors:
T A Heming; A Bidani
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Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  72     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  1992 Jun 
Date Detail:
Created Date:  1992-08-19     Completed Date:  1992-08-19     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  2140-8     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine, University of Texas Medical Branch, Galveston 77550.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bicarbonates / metabolism
Buffers
Capillaries / metabolism
Carbon Dioxide / metabolism*
Carbonic Anhydrases / metabolism
Hydrogen-Ion Concentration
Kinetics
Lung / blood supply,  metabolism*
Male
Perfusion
Protons
Pulmonary Gas Exchange / physiology*
Rats
Rats, Inbred Strains
Chemical
Reg. No./Substance:
0/Bicarbonates; 0/Buffers; 0/Protons; 124-38-9/Carbon Dioxide; EC 4.2.1.1/Carbonic Anhydrases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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