Document Detail


Inflammation associated with the postprandial lipolysis of triglyceride-rich lipoproteins by lipoprotein lipase.
MedLine Citation:
PMID:  19361351     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although hypertriglyceridemia has repeatedly been implicated as an atherogenic condition, there are conflicting reports concerning the atherogenicity of products released from triglyceride-rich lipoproteins by lipoprotein lipase. The hydrolysis of triglyceride is a normal process by which chylomicrons and very low-density lipoproteins are metabolized and cleared from the circulation, which would suggest a beneficial role for lipoprotein lipase in reducing circulating levels of triglyceride and, therefore, reducing atherosclerotic burden. However, many in vitro studies have shown that lipolysis products such as fatty acids induce vascular cell inflammation, which can initiate or exacerbate atherosclerosis. This review summarizes the results and implications of recent studies on the effects of lipoprotein lipase on vascular inflammation, expanding upon existing controversy among human postprandial studies, animal models, and in vitro experimental models.
Authors:
Laura J Higgins; John C Rutledge
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Current atherosclerosis reports     Volume:  11     ISSN:  1534-6242     ISO Abbreviation:  Curr Atheroscler Rep     Publication Date:  2009 May 
Date Detail:
Created Date:  2009-04-13     Completed Date:  2009-08-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100897685     Medline TA:  Curr Atheroscler Rep     Country:  United States    
Other Details:
Languages:  eng     Pagination:  199-205     Citation Subset:  IM    
Affiliation:
Department of Veterinary Medicine: Pharmacology, Microbiology, and Immunology, University of California, Davis, 4206 VM3A, One Shields Avenue, Davis, CA 95616, USA. ljhiggins@ucdavis.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Atherosclerosis / enzymology*
Humans
Inflammation / enzymology*
Lipoprotein Lipase / metabolism*
Postprandial Period / physiology*
Triglycerides / blood*
Chemical
Reg. No./Substance:
0/Triglycerides; EC 3.1.1.34/Lipoprotein Lipase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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