Document Detail


Induction of a mitosis delay and cell lysis by high-level secretion of mouse alpha-amylase from Saccharomyces cerevisiae.
MedLine Citation:
PMID:  11472949     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Some foreign proteins are produced in yeast in a cell cycle-dependent manner, but the cause of the cell cycle dependency is unknown. In this study, we found that Saccharomyces cerevisiae cells secreting high levels of mouse alpha-amylase have elongated buds and are delayed in cell cycle completion in mitosis. The delayed cell mitosis suggests that critical events during exit from mitosis might be disturbed. We found that the activities of PP2A (protein phosphatase 2A) and MPF (maturation-promoting factor) were reduced in alpha-amylase-oversecreting cells and that these cells showed a reduced level of assembly checkpoint protein Cdc55, compared to the accumulation in wild-type cells. MPF inactivation is due to inhibitory phosphorylation on Cdc28, as a cdc28 mutant which lacks an inhibitory phosphorylation site on Cdc28 prevents MPF inactivation and prevents the defective bud morphology induced by overproduction of alpha-amylase. Our data also suggest that high levels of alpha-amylase may downregulate PPH22, leading to cell lysis. In conclusion, overproduction of heterologous alpha-amylase in S. cerevisiae results in a negative regulation of PP2A, which causes mitotic delay and leads to cell lysis.
Authors:
B D Wang; T T Kuo
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Applied and environmental microbiology     Volume:  67     ISSN:  0099-2240     ISO Abbreviation:  Appl. Environ. Microbiol.     Publication Date:  2001 Aug 
Date Detail:
Created Date:  2001-07-26     Completed Date:  2001-10-25     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  7605801     Medline TA:  Appl Environ Microbiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3693-701     Citation Subset:  IM    
Affiliation:
Institute of Molecular Biology, Academia Sinica, Nankang, Taipei 115, Taiwan. bdwang@gate.sinica.edu.tw
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MeSH Terms
Descriptor/Qualifier:
Animals
CDC28 Protein Kinase, S cerevisiae / genetics,  metabolism
Cell Cycle Proteins / metabolism
Culture Media
DNA, Fungal / analysis
Gene Expression Regulation, Fungal*
Maturation-Promoting Factor / metabolism
Mice
Mitosis / physiology*
Phosphoprotein Phosphatases / metabolism
Protein Phosphatase 2
Recombinant Proteins / metabolism
Saccharomyces cerevisiae / genetics*,  physiology*,  ultrastructure
Saccharomyces cerevisiae Proteins*
alpha-Amylases / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/CDC55 protein, S cerevisiae; 0/Cell Cycle Proteins; 0/Culture Media; 0/DNA, Fungal; 0/Recombinant Proteins; 0/Saccharomyces cerevisiae Proteins; EC 2.7.11.22/CDC28 Protein Kinase, S cerevisiae; EC 2.7.11.22/Maturation-Promoting Factor; EC 3.1.3.16/Phosphoprotein Phosphatases; EC 3.1.3.16/Protein Phosphatase 2; EC 3.2.1.1/alpha-Amylases
Comments/Corrections

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