Document Detail


Individual regulation of different hepatocellular functions during sepsis.
MedLine Citation:
PMID:  1518425     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The purpose of this study was to test the hypothesis that different hepatocellular functions are regulated individually during sepsis. This was done by simultaneously measuring bile production, release of liver transaminases, and synthesis of secreted proteins in perfused livers from control and septic rats. Sepsis was induced by cecal ligation and puncture (CLP); control rats were sham-operated. After 16 hours, livers were perfused in situ, and bile flow, synthesis rates of albumin and alpha 1-acid glycoprotein (a major acute-phase protein in rats), and release of glutamic-oxaloacetic transaminase (GOT) and glutamic-pyruvic transaminase (GPT) into perfusate were determined. Within the same livers, sepsis resulted in a 54% increase in the synthesis of alpha 1-acid glycoprotein and approximately 30% inhibition of albumin synthesis concomitant with 50% lower bile flow. The concentrations of GOT and GPT in the perfusate increased slightly during the experiments, both when control and septic livers were perfused. The maintained tissue levels of adenosine triphosphate (ATP) and the uptake of Evans blue dye by less than 1% of the hepatocytes, although a late test of viability, suggest that both control and septic livers remained viable during perfusion. The results are consistent with the concept that different hepatocellular functions are individually regulated during sepsis. Thus, impairment of certain hepatocellular functions does not necessarily imply generalized liver failure.
Authors:
D von Allmen; P O Hasselgren; T Higashiguchi; J E Fischer
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Metabolism: clinical and experimental     Volume:  41     ISSN:  0026-0495     ISO Abbreviation:  Metab. Clin. Exp.     Publication Date:  1992 Sep 
Date Detail:
Created Date:  1992-10-08     Completed Date:  1992-10-08     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0375267     Medline TA:  Metabolism     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  961-9     Citation Subset:  IM    
Affiliation:
Department of Surgery, University of Cincinnati Medical Center, Cincinnati 45267-0558.
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MeSH Terms
Descriptor/Qualifier:
Adenosine Triphosphate / analysis,  metabolism
Alanine Transaminase / blood
Albumins / metabolism
Animals
Aspartate Aminotransferases / blood
Bacterial Infections / blood,  metabolism*,  physiopathology*
Bile / metabolism
Cecum / surgery
Ligation
Liver / metabolism,  pathology,  physiopathology*
Liver Diseases / metabolism,  pathology,  physiopathology*
Male
Orosomucoid / metabolism
Rats
Rats, Inbred Strains
Time Factors
Transaminases / metabolism
Grant Support
ID/Acronym/Agency:
1R01 DK40644/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Albumins; 0/Orosomucoid; 56-65-5/Adenosine Triphosphate; EC 2.6.1.-/Transaminases; EC 2.6.1.1/Aspartate Aminotransferases; EC 2.6.1.2/Alanine Transaminase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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