Document Detail


Indispensable but insufficient role of renal D-amino acid oxidase in chiral inversion of NG-nitro-D-arginine.
MedLine Citation:
PMID:  20564560     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Unidirectionally chiral inversion of N(G)-nitro-D-arginine (D-NNA) to its L-enantiomer (L-NNA) occurred in rats, and it was blocked markedly (ca. 80%) by renal vascular ligation, and entirely (100%) by the D-amino acid oxidase (DAO) inhibitor sodium benzoate, suggesting that renal DAO is essential for the inversion. However, the doses of sodium benzoate administrated were extremely high (e.g., 400 mg/kg) due to its low potency. It is thus possible that sodium benzoate-mediated blockade of D-NNA inversion might be due to its nonspecific (or non-DAO-related) effects. In addition, after D-NNA was incubated with the pure enzyme of DAO in vitro without tissue homogenates, L-NNA was not produced, even though D-NNA was disposed. We propose that this occurred because D-NNA was first converted to its corresponding alpha-keto acid by DAO and then to L-NNA by transaminase(s); however, there was no direct evidence for this process. The goal of this study is to further elucidate the process of D-NNA chiral inversion both in vivo and in in vitro tissue homogenates by comparing mutant ddY/DAO(-/-) mice that lack DAO activity entirely compared to normal ddY/DAO(+/+) mice and Swiss mice. Furthermore, the ability to produce L-NNA from D-NNA-corresponding alpha-keto acids (N(G)-nitroguanidino-2-oxopentanoic acid) produced by porcine kidney-derived DAO (pkDAO) was also studied in the DAO inhibitor-pretreated rats. We found that D-NNA chiral inversion occurred in Swiss mice and ddY/DAO(+/+) mice both in vivo and in in vitro kidney homogenates, but not in ddY/DAO(-/-) mice, correlated to their DAO activities. The alpha-keto acid (N(G)-nitro-guanidino-2-oxopentanoic acid) from D-NNA was able to produce L-NNA, and subsequent vasoconstriction and pressor responses. These results indicate that the role of renal DAO is indispensible but insufficient for chiral inversion of D-NNA and other neutral and polar D-amino acids, and unidentified aminotransferase(s) are involved in a subsequent mechanism for the process of chiral inversion.
Authors:
Yan-Fei Xin; Xin Li; Bin Hao; Nian Gong; Wen-Qiang Sun; Ryuichi Konno; Yong-Xiang Wang
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Chemistry & biodiversity     Volume:  7     ISSN:  1612-1880     ISO Abbreviation:  Chem. Biodivers.     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-06-30     Completed Date:  2010-10-21     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101197449     Medline TA:  Chem Biodivers     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  1413-23     Citation Subset:  IM    
Affiliation:
King's Lab, School of Pharmacy, Shanghai Jiao Tong University, No. 6 Biomedicine Building (Suite 106), 800 Dongchuan Road, Shanghai 200240, PR China.
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MeSH Terms
Descriptor/Qualifier:
Animals
D-Amino-Acid Oxidase / antagonists & inhibitors,  genetics,  metabolism*
Keto Acids / metabolism
Kidney / enzymology
Liver / enzymology
Male
Mice
Nitroarginine / chemistry,  metabolism*
Rats
Rats, Sprague-Dawley
Sodium Benzoate / pharmacology
Stereoisomerism
Swine
Chemical
Reg. No./Substance:
0/Keto Acids; 2149-70-4/Nitroarginine; 532-32-1/Sodium Benzoate; EC 1.4.3.3/D-Amino-Acid Oxidase

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