Document Detail


Increasing mitochondrial superoxide dismutase abundance leads to impairments in protein quality control and ROS scavenging systems and to lifespan shortening.
MedLine Citation:
PMID:  20080171     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The fungal aging model Podospora anserina contains three superoxide dismutases (SODs) in different cellular compartments. While PaSOD1 represents the Cu/Zn isoform located in the cytoplasm and in the mitochondrial inter-membrane space, PaSOD2 localizes to the perinuclear ER. PaSOD3, a protein with a manganese binding domain and a mitochondrial targeting sequence (MTS) is the mitochondrial SOD. Over-expression of PaSod3 leads to lifespan reduction and increased sensitivity against paraquat and hydrogen peroxide. The negative effects of PaSod3 over-expression correlate with a strong reduction in the abundance of mitochondrial peroxiredoxin, PaPRX1, and the matrix protease PaCLPP disclosing impairments of mitochondrial quality control and ROS scavenging pathways in PaSod3 over-expressors. Deletion of PaSod3 leads to increased paraquat sensitivity while hydrogen peroxide sensitivity and lifespan are not significantly changed when compared to the wild-type strain. These latter characteristics are unexpected and challenge the 'mitochondrial free radical theory of aging'.
Authors:
Sandra Zintel; Denise Schwitalla; Karin Luce; Andrea Hamann; Heinz D Osiewacz
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-01-18
Journal Detail:
Title:  Experimental gerontology     Volume:  45     ISSN:  1873-6815     ISO Abbreviation:  Exp. Gerontol.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-06-01     Completed Date:  2010-09-14     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0047061     Medline TA:  Exp Gerontol     Country:  England    
Other Details:
Languages:  eng     Pagination:  525-32     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2010 Elsevier Inc. All rights reserved.
Affiliation:
Institute of Molecular Biosciences and Cluster of Excellence Macromolecular Complexes, Department of Biosciences, J.W. Goethe-University, Max-von-Laue-Strasse 9, Frankfurt am Main, Germany.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Base Sequence
DNA, Fungal / genetics
Free Radical Scavengers / metabolism
Fungal Proteins / genetics,  metabolism
Gene Expression
Genes, Fungal
Green Fluorescent Proteins / genetics,  metabolism
Hydrogen Peroxide / pharmacology
Mitochondria / metabolism*
Models, Biological
Mutation
Paraquat / pharmacology
Podospora / drug effects,  genetics,  metabolism*
Reactive Oxygen Species / metabolism
Recombinant Fusion Proteins / genetics,  metabolism
Superoxide Dismutase / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/DNA, Fungal; 0/Free Radical Scavengers; 0/Fungal Proteins; 0/Reactive Oxygen Species; 0/Recombinant Fusion Proteins; 147336-22-9/Green Fluorescent Proteins; 4685-14-7/Paraquat; 7722-84-1/Hydrogen Peroxide; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Abrupt Telomere Shortening in normal human fibroblasts.
Next Document:  Novel modulators of senescence, aging, and longevity: small non-coding RNAs enter the stage.