Document Detail


Increased transsulfuration mediates longevity and dietary restriction in Drosophila.
MedLine Citation:
PMID:  21930912     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The mechanisms through which dietary restriction enhances health and longevity in diverse species are unclear. The transsulfuration pathway (TSP) is a highly conserved mechanism for metabolizing the sulfur-containing amino acids, methionine and cysteine. Here we show that Drosophila cystathionine β-synthase (dCBS), which catalyzes the rate-determining step in the TSP, is a positive regulator of lifespan in Drosophila and that the pathway is required for the effects of diet restriction on animal physiology and lifespan. dCBS activity was up-regulated in flies exposed to reduced nutrient conditions, and ubiquitous or neuron-specific transgenic overexpression of dCBS enhanced longevity in fully fed animals. Inhibition of the TSP abrogated the changes in lifespan, adiposity, and protein content that normally accompany diet restriction. RNAi-mediated knockdown of dCBS also limited lifespan extension by diet. Diet restriction reduced levels of protein translation in Drosophila, and we show that this is largely caused by increased metabolic commitment of methionine cycle intermediates to transsulfuration. However, dietary supplementation of methionine restored normal levels of protein synthesis to restricted animals without affecting lifespan, indicating that global reductions in translation alone are not required for diet-restriction longevity. Our results indicate a mechanism by which dietary restriction influences physiology and aging.
Authors:
Hadise Kabil; Omer Kabil; Ruma Banerjee; Lawrence G Harshman; Scott D Pletcher
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-09-19
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  108     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2011 Oct 
Date Detail:
Created Date:  2011-10-05     Completed Date:  2011-12-13     Revised Date:  2013-05-23    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  16831-6     Citation Subset:  IM    
Affiliation:
Department of Molecular and Integrative Physiology and Geriatrics Center, University of Michigan, Ann Arbor, MI 48109, USA. hadisekabil@gmail.com
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animal Nutritional Physiological Phenomena / physiology*
Animals
Blotting, Western
Caloric Restriction
Chromatography, High Pressure Liquid
Cloning, Molecular
Computational Biology
Cystathionine beta-Synthase / genetics,  metabolism*
Cysteine / metabolism*
DNA Primers / genetics
Drosophila / physiology*
Energy Intake / physiology*
Gene Expression Regulation, Enzymologic / physiology
Glutathione / metabolism
Longevity / genetics,  physiology*
Methionine / metabolism*
Polymerase Chain Reaction
Protein Biosynthesis / physiology
RNA Interference
Triglycerides / metabolism
Grant Support
ID/Acronym/Agency:
HL58984/HL/NHLBI NIH HHS; P30-AG-013283/AG/NIA NIH HHS; R01AG023166/AG/NIA NIH HHS; R01AG030593/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/DNA Primers; 0/Triglycerides; 52-90-4/Cysteine; 63-68-3/Methionine; 70-18-8/Glutathione; EC 4.2.1.22/Cystathionine beta-Synthase
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Nongenomic glucocorticoid receptor action regulates gap junction intercellular communication and neu...
Next Document:  Forkhead factor FoxO1 is essential for placental morphogenesis in the developing embryo.