Document Detail


Increased renal dopamine and acute renal adaptation to a high-phosphate diet.
MedLine Citation:
PMID:  21325500     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The current experiments explore the role of dopamine in facilitating the acute increase in renal phosphate excretion in response to a high-phosphate diet. Compared with a low-phosphate (0.1%) diet for 24 h, mice fed a high-phosphate (1.2%) diet had significantly higher rates of phosphate excretion in the urine associated with a two- to threefold increase in the dopamine content of the kidney and in the urinary excretion of dopamine. Animals fed a high-phosphate diet had a significant increase in the abundance and activity of renal DOPA (l-dihydroxyphenylalanine) decarboxylase and significant reductions in renalase, monoamine oxidase A, and monoamine oxidase B. The activity of protein kinase A and protein kinase C, markers of activation of renal dopamine receptors, were significantly higher in animals fed a high-phosphate vs. a low-phosphate diet. Treatment of rats with carbidopa, an inhibitor of DOPA decarboxylase, impaired adaptation to a high-phosphate diet. These experiments indicate that the rapid adaptation to a high-phosphate diet involves alterations in key enzymes involved in dopamine synthesis and degradation, resulting in increased renal dopamine content and activation of the signaling cascade used by dopamine to inhibit the renal tubular reabsorption of phosphate.
Authors:
Edward J Weinman; Rajatsubhra Biswas; Deborah Steplock; Peili Wang; Yuen-Sum Lau; Gary V Desir; Shirish Shenolikar
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2011-02-16
Journal Detail:
Title:  American journal of physiology. Renal physiology     Volume:  300     ISSN:  1522-1466     ISO Abbreviation:  Am. J. Physiol. Renal Physiol.     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-05-05     Completed Date:  2011-07-01     Revised Date:  2012-05-01    
Medline Journal Info:
Nlm Unique ID:  100901990     Medline TA:  Am J Physiol Renal Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  F1123-9     Citation Subset:  IM    
Affiliation:
Department of Medicine, University of Maryland School of Medicine, Baltimore, 21201, USA. eweinman1440@yahoo.com
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MeSH Terms
Descriptor/Qualifier:
Adaptation, Physiological
Analysis of Variance
Animals
Carbidopa / pharmacology
Cyclic AMP-Dependent Protein Kinases / metabolism
Dopa Decarboxylase / antagonists & inhibitors,  metabolism
Dopamine / metabolism*,  urine
Enzyme Inhibitors / pharmacology
Kidney / drug effects,  metabolism*
Male
Mice
Mice, Inbred C57BL
Monoamine Oxidase / metabolism
Phosphorus, Dietary / administration & dosage,  metabolism*,  urine
Protein Kinase C / metabolism
Rats
Rats, Sprague-Dawley
Signal Transduction
Time Factors
Up-Regulation
Grant Support
ID/Acronym/Agency:
DK081037/DK/NIDDK NIH HHS; DK086402/DK/NIDDK NIH HHS; DK086465/DK/NIDDK NIH HHS; DK55881/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Phosphorus, Dietary; 38821-49-7/Carbidopa; EC 1.4.3.4/Monoamine Oxidase; EC 1.4.3.4./renalase; EC 2.7.11.11/Cyclic AMP-Dependent Protein Kinases; EC 2.7.11.13/Protein Kinase C; EC 4.1.1.-/Dopa Decarboxylase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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