Document Detail


Increased nitrite reductase activity of fetal versus adult ovine hemoglobin.
MedLine Citation:
PMID:  19028797     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Growing evidence indicates that nitrite, NO2-, serves as a circulating reservoir of nitric oxide (NO) bioactivity that is activated during physiological and pathological hypoxia. One of the intravascular mechanisms for nitrite conversion to NO is a chemical nitrite reductase activity of deoxyhemoglobin. The rate of NO production from this reaction is increased when hemoglobin is in the R conformation. Because the mammalian fetus exists in a low-oxygen environment compared with the adult and is exposed to episodes of severe ischemia during the normal birthing process, and because fetal hemoglobin assumes the R conformation more readily than adult hemoglobin, we hypothesized that nitrite reduction to NO may be enhanced in the fetal circulation. We found that the reaction was faster for fetal than maternal hemoglobin or blood and that the reactions were fastest at 50-80% oxygen saturation, consistent with an R-state catalysis that is predominant for fetal hemoglobin. Nitrite concentrations were similar in blood taken from chronically instrumented normoxic ewes and their fetuses but were elevated in response to chronic hypoxia. The findings suggest an augmented nitrite reductase activity of fetal hemoglobin and that the production of nitrite may participate in the regulation of vascular NO homeostasis in the fetus.
Authors:
Arlin B Blood; Mauro Tiso; Shilpa T Verma; Jennifer Lo; Mahesh S Joshi; Ivan Azarov; Lawrence D Longo; Mark T Gladwin; Daniel B Kim-Shapiro; Gordon G Power
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural     Date:  2008-11-21
Journal Detail:
Title:  American journal of physiology. Heart and circulatory physiology     Volume:  296     ISSN:  0363-6135     ISO Abbreviation:  Am. J. Physiol. Heart Circ. Physiol.     Publication Date:  2009 Feb 
Date Detail:
Created Date:  2009-01-27     Completed Date:  2009-03-12     Revised Date:  2010-09-23    
Medline Journal Info:
Nlm Unique ID:  100901228     Medline TA:  Am J Physiol Heart Circ Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  H237-46     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Division of Neonatology, School of Medicine, Loma Linda University, Loma Linda, CA, USA. ablood@llu.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Anoxia / enzymology
Biocatalysis
Chronic Disease
Disease Models, Animal
Dithionite / chemistry
Female
Fetal Blood / enzymology*
Fetal Hypoxia / enzymology
Hemoglobins / chemistry,  metabolism*
Hydrogen-Ion Concentration
Kinetics
Methemoglobin / metabolism
Nitric Oxide / blood*
Nitrite Reductases / blood*
Nitrites / blood*
Oxygen / blood
Pregnancy
Protein Conformation
Sheep
Grant Support
ID/Acronym/Agency:
HD-03807/HD/NICHD NIH HHS; HL-058091/HL/NHLBI NIH HHS; HL-078706/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Hemoglobins; 0/Nitrites; 10102-43-9/Nitric Oxide; 14844-07-6/Dithionite; 7782-44-7/Oxygen; 9008-37-1/Methemoglobin; EC 1.7.-/Nitrite Reductases
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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