Document Detail


Increased circulating adiponectin levels and decreased leptin/soluble leptin receptor ratio throughout puberty in female ballet dancers: association with body composition and the delay in puberty.
MedLine Citation:
PMID:  20145046     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
INTRODUCTION: Ballet dancers (BDs) have a negative energy balance related to physical training that results in alterations in body composition, sexual development, and adipokine secretion. Our aims were to study anthropometric parameters, body composition, and their relationship with adipokines throughout pubertal development. SUBJECTS AND METHODS: We carried out a prospective follow-up study of 22 female Caucasian BDs (Tanner II stage) followed throughout puberty. Nutritional status was determined by measurement of height, weight, and body mass index (BMI). We calculated growth velocity, bone maturity, and body composition at Tanner stages II, III, and V by dual energy X-ray absorptiometry. Circulating leptin, adiponectin, and soluble leptin receptor (sObR) levels were determined. RESULTS: BDs presented a delay in skeletal maturation during puberty, without affectation of final height. Energy intake was deficient according to their physical exercise, and they had a delay of 1 year in the mean age of menarche. Leptin levels were decreased, whereas sObR and adiponectin levels were increased throughout puberty. The percentage of trunk fat, total fat mass, and fat of the extremities was decreased throughout the study period (P<0.01). Lean mass was increased in the lower extremities, and bone mineral density was normal. CONCLUSION: A negative energy balance together with maintained physical exercise induced modifications in body composition in BDs. Changes in leptin and adiponectin levels appear to be more related to total fat content than to BMI. Furthermore, the onset and delayed progress of puberty may be related with an inadequate energy balance due to increased exercise.
Authors:
M A Donoso; M T Mu?oz-Calvo; V Barrios; G Garrido; F Hawkins; J Argente
Related Documents :
20830526 - Current issues regarding treatment of mitochondrial fatty acid oxidation disorders.
21285476 - Does yoga engender fitness in older adults? a critical review.
18927166 - Review on leptin and adiponectin responses and adaptations to acute and chronic exercise.
16807626 - Consequence of resistance training on body composition and coronary artery disease risk.
16937966 - Adaptations in upper-body maximal strength and power output resulting from long-term re...
15193546 - Creation and characterization of a mouse model of mandibular distraction osteogenesis.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-02-09
Journal Detail:
Title:  European journal of endocrinology / European Federation of Endocrine Societies     Volume:  162     ISSN:  1479-683X     ISO Abbreviation:  Eur. J. Endocrinol.     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-04-27     Completed Date:  2010-05-07     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9423848     Medline TA:  Eur J Endocrinol     Country:  England    
Other Details:
Languages:  eng     Pagination:  905-11     Citation Subset:  IM    
Affiliation:
Department of Endocrinology, Hospital Infantil Universitario Ni?o Jes?s, Department of Pediatrics, Universidad Aut?noma, Madrid, Spain.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adiponectin / blood
Adolescent
Body Composition
Child
Dancing / physiology*
Female
Humans
Leptin / blood*
Menarche
Prospective Studies
Puberty / blood*
Puberty, Delayed / blood
Receptors, Leptin / blood*
Chemical
Reg. No./Substance:
0/Adiponectin; 0/Leptin; 0/Receptors, Leptin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Models to predict changes in serum IGF-I and body composition in response to growth hormone (GH) rep...
Next Document:  Acromegaly per se does not increase the risk for coronary artery disease.