Document Detail


Increased lipolysis in adipose tissues is associated with elevation of systemic free fatty acids and insulin resistance in perilipin null mice.
MedLine Citation:
PMID:  20091459     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Elevated plasma levels of free fatty acids (FFAs) are thought to restrict glucose utilization and induce insulin resistance. Plasma FFA concentrations are primarily governed by lipolysis in adipocytes. Perilipin surrounds the lipid droplet in adipocytes and has a dual role in lipolysis regulation. Perilipin null mice studied by two independent laboratories exhibited similar phenotypes of reduced adipose mass and resistance to diet-induced obesity, but have inconsistent metabolic parameters such as plasma levels of FFA, glucose, and insulin. This discrepancy may be due to differences in genetic background, generation, and nutritional status of the animals examined. In this study, we examined the major metabolic parameters in 129/SvEv perilipin null mice fasted for 4 h and observed increased plasma concentrations of FFA, glycerol, glucose, and insulin. An increase in the score for the homeostasis model assessment of insulin resistance index confirmed the insulin resistance in perilipin null mice, which may be attributed to the plasma FFA elevation. Basal lipolysis was increased in adipose tissues or primary adipocytes isolated from perilipin null mice with increased mass and activity of hormone-sensitive lipase and adipose triglyceride lipase. The increased lipolytic action may accelerate FFA efflux from the adipose tissues to the bloodstream, thereby accounting for systemic FFA elevation and, hence, insulin resistance in perilipin null mice.
Authors:
W Zhai; C Xu; Y Ling; S Liu; J Deng; Y Qi; C Londos; G Xu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-01-20
Journal Detail:
Title:  Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et métabolisme     Volume:  42     ISSN:  1439-4286     ISO Abbreviation:  Horm. Metab. Res.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-03-31     Completed Date:  2010-06-28     Revised Date:  2011-10-17    
Medline Journal Info:
Nlm Unique ID:  0177722     Medline TA:  Horm Metab Res     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  247-53     Citation Subset:  IM    
Copyright Information:
Georg Thieme Verlag KG Stuttgart New York.
Affiliation:
Department of Physiology and Pathophysiology, Peking University Health Science Center, Beijing, China.
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MeSH Terms
Descriptor/Qualifier:
Adipocytes / metabolism
Adipose Tissue / cytology,  enzymology,  metabolism*
Animals
Blood Glucose / metabolism
Body Weight
Carboxylic Ester Hydrolases / metabolism
Cell Separation
Cells, Cultured
Fatty Acids, Nonesterified / blood,  metabolism*
Glycerol / blood
Insulin / blood
Insulin Resistance*
Lipolysis*
Mice
Organ Size
Phosphoproteins / deficiency*,  metabolism
Sterol Esterase / metabolism
Triglycerides / blood
Chemical
Reg. No./Substance:
0/Blood Glucose; 0/Fatty Acids, Nonesterified; 0/Phosphoproteins; 0/Triglycerides; 0/perilipin 1; 11061-68-0/Insulin; 56-81-5/Glycerol; EC 3.1.1.-/Carboxylic Ester Hydrolases; EC 3.1.1.1/desnutrin protein, mouse; EC 3.1.1.13/Sterol Esterase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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