Document Detail


Increased Langerhan cell density and corneal nerve damage in diabetic patients: role of immune mechanisms in human diabetic neuropathy.
MedLine Citation:
PMID:  20851037     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
AIM/HYPOTHESIS: Immune mechanisms have been proposed to play a role in the development of diabetic neuropathy. We employed in vivo corneal confocal microscopy (CCM) to quantify the presence and density of Langerhans cells (LCs) in relation to the extent of corneal nerve damage in Bowman's layer of the cornea in diabetic patients.
METHODS: 128 diabetic patients aged 58 ± 1 yrs with a differing severity of neuropathy based on Neuropathy Deficit Score (NDS-4.7 ± 0.28) and 26 control subjects aged 53 ± 3 yrs were examined. Subjects underwent a full neurological evaluation, evaluation of corneal sensation with non-contact corneal aesthesiometry (NCCA) and corneal nerve morphology using corneal confocal microscopy (CCM).
RESULTS: The proportion of individuals with LCs was significantly increased in diabetic patients (73.8%) compared to control subjects (46.1%), P = 0.001. Furthermore, LC density (no/mm(2)) was significantly increased in diabetic patients (17.73 ± 1.45) compared to control subjects (6.94 ± 1.58), P = 0.001 and there was a significant correlation with age (r = 0.162, P = 0.047) and severity of neuropathy (r = -0.202, P = 0.02). There was a progressive decrease in corneal sensation with increasing severity of neuropathy assessed using NDS in the diabetic patients (r = 0.414, P = 0.000). Corneal nerve fibre density (P < 0.001), branch density (P < 0.001) and length (P < 0.001) were significantly decreased whilst tortuosity (P < 0.01) was increased in diabetic patients with increasing severity of diabetic neuropathy.
CONCLUSION: Utilising in vivo corneal confocal microscopy we have demonstrated increased LCs in diabetic patients particularly in the earlier phases of corneal nerve damage suggestive of an immune mediated contribution to corneal nerve damage in diabetes.
Authors:
M Tavakoli; A J M Boulton; N Efron; R A Malik
Related Documents :
10361937 - I-123 mibg cardiac imaging in diabetic neuropathy before and after epalrestat therapy.
3886757 - Clinical and biochemical aspects of chromium deficiency.
16710647 - The restorative effects of pulsed infrared light therapy on significant loss of periphe...
15259837 - Neuroticism, a central link between somatic and psychiatric morbidity: path analysis of...
3325297 - Fasting and l-364,718 prevent cholecystokinin-induced elevations of plasma insulin levels.
18338077 - First-degree relatives of patients with type 2 diabetes mellitus and risk of non-alcoho...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-09-20
Journal Detail:
Title:  Contact lens & anterior eye : the journal of the British Contact Lens Association     Volume:  34     ISSN:  1476-5411     ISO Abbreviation:  Cont Lens Anterior Eye     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2011-01-18     Completed Date:  2011-05-17     Revised Date:  2012-02-02    
Medline Journal Info:
Nlm Unique ID:  9712714     Medline TA:  Cont Lens Anterior Eye     Country:  England    
Other Details:
Languages:  eng     Pagination:  7-11     Citation Subset:  IM    
Copyright Information:
Crown Copyright © 2010. Published by Elsevier Ltd. All rights reserved.
Affiliation:
Division of Cardiovascular Medicine, University of Manchester and Manchester Royal Infirmary, Manchester, M13 9NT, UK.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Cell Count
Cornea / immunology*,  innervation*,  pathology
Diabetic Neuropathies / immunology*,  pathology*
Female
Humans
Langerhans Cells / immunology*,  pathology*
Male
Middle Aged
Grant Support
ID/Acronym/Agency:
1 R01 NS46259-01/NS/NINDS NIH HHS; R01 NS046259-01/NS/NINDS NIH HHS
Comments/Corrections
Comment In:
Cont Lens Anterior Eye. 2011 Apr;34(2):98; author reply 99   [PMID:  21215678 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  The roots of a new green revolution.
Next Document:  Cytokinin signaling and transcriptional networks.