Document Detail


Incongruent follicular development reduces gamete intrafallopian transfer pregnancy rate.
MedLine Citation:
PMID:  8941066     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: To test the null hypothesis that asymmetric or incongruent follicular development does not influence GIFT pregnancy rates (PRs). DESIGN: A prospective comparative study. SETTING: Tertiary referral center for infertility. PATIENT(S): Ninety-three consecutive infertile patients having GIFT. INTERVENTION(S): Controlled ovarian hyperstimulation and GIFT. MAIN OUTCOME MEASURE(S): The incongruity ratio as a parameter of the asymmetry in follicular development and the clinical PR. RESULT(S): An inverse relationship was observed between the degree of incongruity and the clinical PRs. In patients with an incongruity ratio < 1.5, the clinical PR was 56.1%. In patients with an incongruity ratio > or = 1.5, the clinical PR was only 17.3%. CONCLUSION(S): Incongruent follicular development during controlled ovarian hyperstimulation has a marked negative influence on the outcome of GIFT cycles. Factors such as side of the gamete transfer and oocyte quality did not account for the observed differences.
Authors:
L Rombauts; D Webster; C E Wood; D L Healy
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Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  Fertility and sterility     Volume:  66     ISSN:  0015-0282     ISO Abbreviation:  Fertil. Steril.     Publication Date:  1996 Dec 
Date Detail:
Created Date:  1997-01-02     Completed Date:  1997-01-02     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0372772     Medline TA:  Fertil Steril     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  987-90     Citation Subset:  IM    
Affiliation:
Monash IVF, Epworth Hospital, Richmond, Victoria, Australia.
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MeSH Terms
Descriptor/Qualifier:
Adult
Female
Gamete Intrafallopian Transfer*
Humans
Ovarian Follicle / physiology*,  ultrasonography
Ovary / physiopathology
Pregnancy Rate*
Prospective Studies
Stimulation, Chemical

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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