Document Detail


In vivo covalent binding of clofibric acid to human plasma proteins and rat liver proteins.
MedLine Citation:
PMID:  1930265     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recent studies have shown that acyl-glucuronide conjugates are chemically reactive electrophilic metabolites that can undergo transacylation reactions resulting in intra-molecular rearrangement, hydrolysis and covalent binding of aglycone to albumin both in vitro and in vivo. The hypolipidaemic agent clofibrate is eliminated almost entirely as clofibric acid glucuronide in humans and rats. The formation of clofibric acid-protein adducts was investigated in 14 patients receiving 0.5-2.0 g/day of clofibrate for hypercholesterolaemia, and in liver homogenates from 20 rats administered 280 mg/kg/day of clofibric acid for up to 21 days. Total clofibric acid concentrations in the patients ranged from 0 to 114 mg/L. Covalently bound clofibric acid-protein adducts were detected in all patients, even in one subject in whom there was no measurable plasma clofibric acid. Concentrations ranged from 2.2 to 53.4 ng/mg protein and, in eight patients receiving 1.0 g/day of clofibrate, were correlated (P less than 0.05) with renal function as assessed by creatinine clearance. Clofibric acid-protein adducts were also present in rat liver homogenates, and increased with increasing duration of treatment (P less than 0.0001), from a mean (SE) of 10.1 (0.7) to 32.3 (1.6) ng/mg protein. The covalent binding of drugs to tissue macromolecules has traditionally been associated with toxicity. Further research is required to elucidate the role of acyl-glucuronide conjugates in the formation of drug-protein adducts and their biological consequences.
Authors:
B C Sallustio; K M Knights; B J Roberts; R Zacest
Related Documents :
8912685 - Up-regulation of the expression of the gene for liver fatty acid-binding protein by lon...
9048555 - The sterol carrier protein-2 fatty acid binding site: an nmr, circular dichroic, and fl...
9187305 - Fatty acid binding proteins reduce 15-lipoxygenase-induced oxygenation of linoleic acid...
2040395 - Prevention of inhibitory effect of free fatty acids on insulin binding and action in is...
4134135 - Effect of pipecolic acid isomers on the biogenesis of actinomycins.
7030915 - Uptake, metabolism and excretion of orally and intravenously administered, 14c- and 3h-...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Biochemical pharmacology     Volume:  42     ISSN:  0006-2952     ISO Abbreviation:  Biochem. Pharmacol.     Publication Date:  1991 Sep 
Date Detail:
Created Date:  1991-10-31     Completed Date:  1991-10-31     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  0101032     Medline TA:  Biochem Pharmacol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  1421-5     Citation Subset:  IM    
Affiliation:
Department of Clinical Pharmacology, Queen Elizabeth Hospital, Woodville South, Australia.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Blood Proteins / metabolism*
Clofibric Acid / blood,  pharmacokinetics*,  therapeutic use
Creatinine / blood
Glucuronates / metabolism,  urine
Humans
Hypercholesterolemia / drug therapy
Liver / metabolism*
Male
Proteins / metabolism*
Rats
Regression Analysis
Chemical
Reg. No./Substance:
0/Blood Proteins; 0/Glucuronates; 0/Proteins; 60-27-5/Creatinine; 882-09-7/Clofibric Acid

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  The flavin-containing monooxygenase of mouse kidney. A comparison with the liver enzyme.
Next Document:  The role of protein kinase C and the phosphatidylinositol cycle in multidrug resistance in human ova...