Document Detail


In vitro inhibition of aggrecanase activity by tetracyclines and proteoglycan loss from osteoarthritic human articular cartilage.
MedLine Citation:
PMID:  20069635     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Tetracyclines were reported to slow down the progression of cartilage damage both in an animal model of osteoarthritis (OA) and in humans. In search for the underlying mechanisms we examined whether tetracyclines possess an inhibitory potential on the activity of aggrecanases and inflammatory mediators and can thus prevent proteoglycan (PG) loss from human articular cartilage. In vitro activity of aggrecanase-1 and -2 was recorded in the presence of 1-100 microM tetracycline, minocycline, or doxycyline. Human knee articular cartilage explants were sorted according to the degree of OA and treated for 10 days with tetracycline derivatives in the presence of interleukin-1 (IL-1beta). Synthesis and loss of PGs, nitric oxide (NO), and prostaglandin E(2) (PGE(2)), as well as the viability were determined. Tetracyclines derivatives dose-dependently inhibited the activities of both aggrecanases in vitro, whereas no inhibitory effect of tetracyclines on any proteoglycanolytic activities within IL-1beta-treated human cartilage explants were found. Tetracyclines can significantly modulate NO and PGE(2) levels, but have no effect on PG synthesis and loss within the same human cartilage explant cultures. Altogether, our data show that tetracyclines have no inhibitory potential on any proteoglycanolytic activities within mild or moderately affected human OA cartilage at therapeutic achievable plasma levels.
Authors:
J?rgen Steinmeyer; Jens Kordelle; Henning St?rz
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of orthopaedic research : official publication of the Orthopaedic Research Society     Volume:  28     ISSN:  1554-527X     ISO Abbreviation:  J. Orthop. Res.     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-04-21     Completed Date:  2010-05-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8404726     Medline TA:  J Orthop Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  828-33     Citation Subset:  IM    
Copyright Information:
(c) 2010 Orthopaedic Research Society. Published by Wiley Periodicals, Inc.
Affiliation:
Department of Orthopaedic Surgery, University Hospital Giessen and Marburg GmbH, Giessen, Germany. juergen.steinmeyer@ortho.med.uni-giessen.de
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MeSH Terms
Descriptor/Qualifier:
ADAM Proteins / antagonists & inhibitors*
Aged
Cartilage, Articular / metabolism*
Cells, Cultured
Chondrocytes / cytology,  drug effects
Dinoprostone / biosynthesis
Humans
Middle Aged
Nitric Oxide / biosynthesis
Osteoarthritis / drug therapy*,  metabolism
Procollagen N-Endopeptidase / antagonists & inhibitors*
Protease Inhibitors / pharmacology*
Proteoglycans / metabolism*
Tetracyclines / pharmacology*,  therapeutic use
Chemical
Reg. No./Substance:
0/Protease Inhibitors; 0/Proteoglycans; 0/Tetracyclines; 10102-43-9/Nitric Oxide; 363-24-6/Dinoprostone; EC 3.4.24.-/ADAM Proteins; EC 3.4.24.-/ADAMTS5 protein, human; EC 3.4.24.-/aggrecanase-1; EC 3.4.24.14/Procollagen N-Endopeptidase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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