Document Detail


Immunomodulatory effect of pentoxifylline in suppressing experimental autoimmune myocarditis.
MedLine Citation:
PMID:  12030348     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although a recent clinical study reported the beneficial effects of pentoxifylline (PTX), a phosphodiesterase inhibitor, on both symptoms and cardiac function in dilated cardiomyopathy (DCM), the precise mechanism of the drug has not been delineated. This study examined the efficacy of PTX in the treatment of experimental autoimmune myocarditis (EAM), as a model of the autoimmune mechanism involved in DCM. Oral PTX, or saline as control, was administered to Lewis rats at 150mg/kg body weight per day bid daily from 5 days before immunization with cardiac myosin until death on Day 21. Histological examination of the hearts showed PTX significantly reduced the severity of EAM. mRNA expression of tumor necrosis factor (TNF)-alpha, interleukin (IL)-4, IL-6, and IL-10 was significantly reduced in peripheral blood mononuclear cells, but expression of IL-4 and IL-6 was upregulated in heart tissue. PTX in vitro could suppress T cell proliferation and inhibit TNF-alpha and interferon-gamma production. In conclusion, the immunomodulatory effects of PTX had a significant therapeutic result in EAM. This is the first report to describe such an effect of PTX in a specific animal model for DCM.
Authors:
Hitoshi Takehana; Takayuki Inomata; Hiroe Niwano; Mototsugu Nishii; Chieko Matsuda; Ken Kohno; Yoji Machida; Tohru Izumi
Related Documents :
8796378 - Endotoxemia and complement activation after severe burn injuries--effects on leukocytes...
8733758 - Anorexia induced by activators of the signal transducer gp 130.
9408298 - Relevance of cytokine production to infected pancreatic necrosis.
18596918 - Six years follow-up of the levels of tnf-alpha and il-6 in patients with complex region...
234788 - Treatment of an established graft-versus-host reaction in akr mice by adoptive immunoth...
10385398 - Insulin-like growth factor i protects oligodendrocytes from tumor necrosis factor-alpha...
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Circulation journal : official journal of the Japanese Circulation Society     Volume:  66     ISSN:  1346-9843     ISO Abbreviation:  Circ. J.     Publication Date:  2002 May 
Date Detail:
Created Date:  2002-05-27     Completed Date:  2002-11-06     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  101137683     Medline TA:  Circ J     Country:  Japan    
Other Details:
Languages:  eng     Pagination:  499-504     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine and Cardiology, Kitasato University School of Medicine, Sagamihara, Kanagawa, Japan. takehana@med.kitasato-u.ac.jp
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adjuvants, Immunologic / therapeutic use*
Animals
Autoimmune Diseases / drug therapy*,  immunology,  metabolism,  pathology
Cell Division / drug effects
Cytokines / biosynthesis
Female
Myocarditis / drug therapy*,  immunology,  metabolism,  pathology
Myocardium / metabolism
Pentoxifylline / therapeutic use*
Rats
Rats, Inbred Lew
Severity of Illness Index
T-Lymphocytes / immunology,  pathology
Chemical
Reg. No./Substance:
0/Adjuvants, Immunologic; 0/Cytokines; 6493-05-6/Pentoxifylline

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Head-to-head comparison of fundamental, tissue harmonic and contrast harmonic imaging with or withou...
Next Document:  Afterdepolarizations promote the transition from ventricular tachycardia to fibrillation in a three-...