Document Detail


Immunomodulation of monocyte-derived dendritic cells through ligation of tumor-produced mucins to Siglec-9.
MedLine Citation:
PMID:  20971061     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Dendritic cells (DCs) play an essential role in the induction and maintenance of an effective immune response and express multiple siglecs. In the present study, we investigated whether or not the ligation of tumor-produced mucins with Siglec-9 expressed on immature DCs is related to escape from immunosurveillance in the tumor-bearing state. Expression of Siglec-9 was up-regulated on the development of monocytes into immature DCs and was decreased in mature DCs. Binding of various mucins and artificial glycopolymers carrying poly (NeuAc α2,6 LacNAc) or poly (NeuAc α2,3 LacNAc) to Siglec-9 was demonstrated by means of a plate assay. These mucins also bound to the surface of immature DCs. When immature DCs were treated with LPS in the presence of these mucins or artificial glycopolymers, the production of IL-12 was significantly reduced, but that of IL-10 was not. Furthermore, IL-12 production was decreased to a similar level on treatment with anti-Siglec-9 mAb. Mucins prepared from serum of cancer patients actually could bind to Siglec-9. These results suggest that Siglec-9 expressed on DCs is involved in immunoregulation through ligation with mucins in an epithelial cancer patient.
Authors:
Mariko Ohta; Akiko Ishida; Munetoyo Toda; Kaoru Akita; Mizue Inoue; Keishi Yamashita; Masashi Watanabe; Takeomi Murata; Taichi Usui; Hiroshi Nakada
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-10-29
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  402     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-11-29     Completed Date:  2011-01-13     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  663-9     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Molecular Biosciences, Faculty of Life Sciences, Kyoto Sangyo University, Kita-ku, Kyoto, Japan.
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MeSH Terms
Descriptor/Qualifier:
Antigens, CD / metabolism*
Carcinoma / immunology*
Cell Line, Tumor
Dendritic Cells / immunology*
Humans
Immunomodulation
Lectins / metabolism*
Monocytes / immunology*
Mucins / blood,  metabolism*
Neoplasms / immunology*
Tumor Escape*
Chemical
Reg. No./Substance:
0/Antigens, CD; 0/Lectins; 0/Mucins; 0/SIGLEC9 protein, human

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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