Document Detail


Immunohistochemical analysis of IQGAP1 expression in human colorectal carcinomas: its overexpression in carcinomas and association with invasion fronts.
MedLine Citation:
PMID:  11790459     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In histopathological sections, it is frequently observed that carcinoma cells move in and invade the stroma as coherent cell nests, rather than single cells. We have called this type of movement 'cohort migration (CM)' and developed an in vitro model, in which human colon adenocarcinoma cells move as coherent cell sheets when stimulated with naturally occurring motogenic factor, hepatocyte growth factor/scatter factor (HGF/SF). In this CM model, localized release from cell-cell adhesion is essential for cell movement. Recently, we have shown that IQGAP1, a target molecule of Cdc42 and Rac1 small GTPases, is involved in this localized release from the E-cadherin-based cell-cell adhesion during CM. In this study, we examined expression of IQGAP1 immunohistochemically in human colorectal tissues and found that IQGAP1 was overexpressed in carcinoma tissues compared with normal counterparts. Within the carcinoma tissue, IQGAP1 tended to be expressed more at the invasion front than at the upper portions, and higher levels of expression were observed in deeper two-thirds of carcinoma tissues than in the superficial one-third. This expression pattern showing stronger signals in deeper portions was most apparent in advanced carcinomas that invaded into the subserosa. These findings supported a role of IQGAP1 in colon carcinoma invasion.
Authors:
Kazuki Nabeshima; Yoshiya Shimao; Teruhiko Inoue; Masashi Koono
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Cancer letters     Volume:  176     ISSN:  0304-3835     ISO Abbreviation:  Cancer Lett.     Publication Date:  2002 Feb 
Date Detail:
Created Date:  2002-01-15     Completed Date:  2002-03-05     Revised Date:  2005-08-12    
Medline Journal Info:
Nlm Unique ID:  7600053     Medline TA:  Cancer Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  101-9     Citation Subset:  IM    
Affiliation:
Department of Pathology, Miyazaki Medical College Hospital, 5200 Kihara, Kiyotake, Miyazaki 889-1692, Japan. kaznabes@post.miyazaki-med.ac.jp
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MeSH Terms
Descriptor/Qualifier:
Adenocarcinoma / metabolism
Adult
Aged
Aged, 80 and over
Cadherins / metabolism
Carcinoma / metabolism*
Carrier Proteins / biosynthesis*
Cell Adhesion
Cell Movement
Colon / metabolism
Colorectal Neoplasms / metabolism*
Female
Gene Expression Regulation, Neoplastic
Humans
Immunohistochemistry
Male
Middle Aged
Neoplasm Invasiveness
Tumor Cells, Cultured
ras GTPase-Activating Proteins*
Chemical
Reg. No./Substance:
0/Cadherins; 0/Carrier Proteins; 0/IQ motif containing GTPase activating protein 1; 0/ras GTPase-Activating Proteins

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