Document Detail


Autoimmunity, autoinflammation and lymphoma in combined immunodeficiency (CID).
MedLine Citation:
PMID:  20146941     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
A number of primary immunodeficiencies are associated with autoimmune phenomena, e.g. Wiskott-Aldrich Syndrome, Common Variable Immunodeficiency and Hyper-IgM Syndrome. The common denominator is a dysregulation of immune responses affecting T and B cells with central and/or peripheral tolerance mechanisms being disturbed. Autoimmunity and autoinflammation may also occur in atypical phenotypes of combined immunodeficiencies (CID) usually associated with severe infectious complications. These unexpected presentations of classical CID are very instructive in how low numbers of T and B cells go hand in hand with skewing of lymphoid repertoires and function. The resulting immune dysregulation may lead to self-reactivity with organ damage and malignancy.
Authors:
Catharina Schuetz; Tim Niehues; Wilhelm Friedrich; Klaus Schwarz
Publication Detail:
Type:  Journal Article; Review     Date:  2010-02-08
Journal Detail:
Title:  Autoimmunity reviews     Volume:  9     ISSN:  1873-0183     ISO Abbreviation:  Autoimmun Rev     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-05-11     Completed Date:  2010-09-20     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101128967     Medline TA:  Autoimmun Rev     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  477-82     Citation Subset:  IM    
Copyright Information:
2010 Elsevier B.V. All rights reserved.
Affiliation:
Department of Pediatrics and Adolescent Medicine, University Hospital Ulm, Germany. catharina.schuetz@uniklinik-ulm.de
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MeSH Terms
Descriptor/Qualifier:
Animals
Autoantigens / immunology*
Autoimmunity
B-Lymphocytes / immunology
Humans
Immune Tolerance
Inflammation
Lymphoma / complications,  immunology*
Severe Combined Immunodeficiency / complications,  immunology*
T-Lymphocytes / immunology
Chemical
Reg. No./Substance:
0/Autoantigens

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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